Abstract
High intrathecal levels of anti-myelin basic protein (MBP) IgM were previously found to be significantly associated with early favorable course in a cohort of patients with multiple sclerosis (MS). A mAb to MBP 105-120 recognizing the 222-228 epitope of the extracellular domain of high affinity immunoglobulin gamma Fc-receptor I (CD64) was isolated from EBV(+) B cell clones of long-term stable RRMS patients. This mAb exerted immunosuppressive activity on MS-derived T cell lines through induction and release of high amounts of interleukin-10 and decreased levels of interleukin-12 from activated monocytes providing the biological basis for a potential new treatment for MS and other immune-mediated neurological disorders.
Copyright © 2013 Elsevier B.V. All rights reserved.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Adult
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Antibodies, Monoclonal / blood
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Antibodies, Monoclonal / pharmacology*
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B-Lymphocytes / drug effects
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B-Lymphocytes / immunology
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B-Lymphocytes / metabolism
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Cell Line
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Cell Proliferation / drug effects
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Cohort Studies
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Cytokines / genetics
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Cytokines / metabolism
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DNA-Binding Proteins / immunology
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Enzyme-Linked Immunosorbent Assay
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Epitope Mapping
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Female
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Flow Cytometry
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Humans
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Immunoglobulin G / blood
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Immunoglobulin M / blood
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Immunoprecipitation
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Immunosuppressive Agents / blood
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Immunosuppressive Agents / pharmacology*
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Male
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Middle Aged
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Multiple Sclerosis / pathology*
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Myelin Basic Protein / immunology*
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Myelin-Oligodendrocyte Glycoprotein / chemistry
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Myelin-Oligodendrocyte Glycoprotein / immunology
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Peptides / immunology
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RNA, Messenger / metabolism
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Receptors, IgG / genetics
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Receptors, IgG / metabolism*
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Statistics, Nonparametric
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T-Lymphocytes / drug effects*
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T-Lymphocytes / immunology
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T-Lymphocytes / metabolism
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Transcription Factors / immunology
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Transfection
Substances
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Antibodies, Monoclonal
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Cytokines
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DNA-Binding Proteins
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Immunoglobulin G
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Immunoglobulin M
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Immunosuppressive Agents
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MYT1 protein, human
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Myelin Basic Protein
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Myelin-Oligodendrocyte Glycoprotein
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Peptides
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RNA, Messenger
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Receptors, IgG
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Transcription Factors