Estrogen Receptor α (ERα) and Estrogen Related Receptor α (ERRα) are both transcriptional regulators of the Runx2-I isoform

Mol Cell Endocrinol. 2013 Apr 30;369(1-2):150-60. doi: 10.1016/j.mce.2013.01.024. Epub 2013 Feb 8.

Abstract

Runx2 is a master regulator of bone development and has also been described as an oncogene. Estrogen Receptor α (ERα) and Estrogen Related Receptor α (ERRα), both implicated in bone metabolism and breast cancer, have been shown to share common transcriptional targets. Here, we show that ERα is a positive regulator of Runx2-I transcription. Moreover, ERRα can act as a transcriptional activator of Runx2-I in presence of peroxisome proliferator activated receptor gamma coactivator-1 alpha (PGC-1α). In contrast, ERRα behaves as a negative regulator of Runx2-I transcription in presence of PGC-1β. ERα and ERRα cross-talk via a common estrogen receptor response element on the Runx2-I promoter. In addition, estrogen regulates PGC-1β that in turn is able to modulate both ERα and ERRα transcriptional activity.

MeSH terms

  • Breast Neoplasms / genetics
  • Breast Neoplasms / metabolism
  • Cell Line, Tumor
  • Core Binding Factor Alpha 1 Subunit / genetics*
  • Core Binding Factor Alpha 1 Subunit / metabolism
  • ERRalpha Estrogen-Related Receptor
  • Estrogen Receptor alpha / genetics
  • Estrogen Receptor alpha / physiology*
  • Female
  • Gene Expression Regulation
  • HeLa Cells
  • Humans
  • MCF-7 Cells
  • Models, Genetic
  • Promoter Regions, Genetic
  • Protein Isoforms / genetics
  • Protein Isoforms / metabolism
  • Receptors, Estrogen / genetics
  • Receptors, Estrogen / physiology*

Substances

  • Core Binding Factor Alpha 1 Subunit
  • Estrogen Receptor alpha
  • Protein Isoforms
  • RUNX2 protein, human
  • Receptors, Estrogen