The role of hematopoietic stem cell transplantation in SP110 associated veno-occlusive disease with immunodeficiency syndrome

Pediatr Allergy Immunol. 2013 May;24(3):250-6. doi: 10.1111/pai.12051. Epub 2013 Mar 1.

Abstract

Background: Veno-occlusive disease with immunodeficiency (VODI) is an autosomal recessive disorder of combined immunodeficiency (CID) and hepatic injury. Hematopoietic stem cell transplantation (HSCT) - the only definitive treatment for CID - appeared to have a high rate of complications in a previous report. In this study, we describe a new group of patients with VODI highlighting further clinical and immunologic aspects of this disease and re-evaluating the effectiveness of HSCT for the treatment of this disorder.

Patients and methods: Review of clinical data, immunologic features, molecular studies, treatment, and final outcome of eight kindred members with VODI.

Results: The patients described had clinical and immunologic findings consistent with VODI. The molecular studies revealed a new mutation in the SP110 gene. HSCT was carried out in five patients and was successful in three.

Conclusions: The diagnosis of VODI should be considered in all patients regardless of ethnicity with a severe combined immunodeficiency (SCID)-like presentation, especially with a normal mitogen response, or with signs of hepatic injury. VODI is a primary immune deficiency, which can be successfully corrected by bone marrow transplantation if applied early in the course of disease using appropriate conditioning.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Child, Preschool
  • DNA Mutational Analysis
  • Hematopoietic Stem Cell Transplantation*
  • Hepatic Veno-Occlusive Disease / complications
  • Hepatic Veno-Occlusive Disease / genetics
  • Hepatic Veno-Occlusive Disease / therapy*
  • Humans
  • Immunologic Deficiency Syndromes / complications
  • Immunologic Deficiency Syndromes / genetics
  • Immunologic Deficiency Syndromes / therapy*
  • Infant
  • Infant, Newborn
  • Liver / pathology
  • Male
  • Minor Histocompatibility Antigens
  • Nuclear Proteins / genetics
  • Transplantation Conditioning
  • Treatment Outcome

Substances

  • Minor Histocompatibility Antigens
  • Nuclear Proteins
  • Sp110 protein, human