Abstract
Receptor Interacting Protein 1 (RIP1) kinase is one of the key mediators of tumor necrosis factor alpha (TNF-α) signaling and is critical for activation of necroptotic cell death. We developed a method for expression of recombinant kinase, utilizing baculovirus co-infection of Cdc37, an Hsp90 co-chaperone, and RIP1-His, followed by a two-step purification scheme. After optimization, 1-3mg of highly purified RIP1 kinase was typically obtained from a 1L of Sf9 cells. The recombinant protein displayed kinase activity that was blocked by RIP1 inhibitors, necrostatins. The purified protein was used to develop a simple and robust thermal shift assay for further assessment of RIP1 inhibitors.
Copyright © 2013 Elsevier Inc. All rights reserved.
Publication types
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Research Support, N.I.H., Extramural
MeSH terms
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Animals
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Baculoviridae / genetics*
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Cell Line
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Cloning, Molecular*
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Humans
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Imidazoles / pharmacology
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Indoles / pharmacology
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Protein Kinase Inhibitors / pharmacology
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Receptor-Interacting Protein Serine-Threonine Kinases / antagonists & inhibitors
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Receptor-Interacting Protein Serine-Threonine Kinases / genetics*
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Receptor-Interacting Protein Serine-Threonine Kinases / isolation & purification
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Receptor-Interacting Protein Serine-Threonine Kinases / metabolism
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Recombinant Proteins / genetics
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Recombinant Proteins / isolation & purification
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Recombinant Proteins / metabolism
Substances
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Imidazoles
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Indoles
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Protein Kinase Inhibitors
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Recombinant Proteins
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necrostatin-1
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RIPK1 protein, human
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Receptor-Interacting Protein Serine-Threonine Kinases