Lipopolysaccharide (LPS) alters phosphatidylcholine metabolism in elicited peritoneal macrophages

J Leukoc Biol. 1990 Jul;48(1):38-42. doi: 10.1002/jlb.48.1.38.

Abstract

We investigated the effects of LPS on mouse peritoneal macrophage phospholipids using radiolabeled precursors. LPS (200 ng/ml) stimulated incorporation of [32P] into all classes of phospholipids within 0.5 hr, and after 2 hr the increase was 60% greater than controls. Separation of the phospholipid classes by thin-layer chromatography revealed a selective increase in incorporation of label into phosphatidylcholine (PC) (90% increase compared to approximately 50% in the other phospholipids). In macrophages labeled with [3H]-choline, LPS stimulated both the incorporation of label into PC and the release of incorporated label into the medium. The time dependencies of stimulated [3H] release and [32P] incorporation were similar. These data are consistent with the hypothesis that LPS activates macrophages via a PC-specific phospholipase-dependent mechanism.

MeSH terms

  • Animals
  • Cells, Cultured
  • Choline / metabolism
  • Female
  • Lipopolysaccharides / pharmacology*
  • Macrophages / drug effects
  • Macrophages / metabolism*
  • Mice
  • Peritoneal Cavity / cytology*
  • Phosphatidylcholines / metabolism*
  • Phospholipases / physiology
  • Phosphorus Radioisotopes
  • Time Factors
  • Tritium

Substances

  • Lipopolysaccharides
  • Phosphatidylcholines
  • Phosphorus Radioisotopes
  • Tritium
  • Phospholipases
  • Choline