Boerhaavia diffusa L. attenuates angiotensin II-induced hypertrophy in H9c2 cardiac myoblast cells via modulating oxidative stress and down-regulating NF-κβ and transforming growth factor β1

Br J Nutr. 2013 Oct;110(7):1201-10. doi: 10.1017/S0007114513000561. Epub 2013 Apr 16.

Abstract

The present study evaluated the antihypertrophic potential of the ethanolic extract of Boerhaavia diffusa (BDE), a well-known edible cardiotonic plant reported in Ayurveda against angiotensin II-induced hypertrophy in H9c2 cardiac myoblast cells. Markers of hypertrophy such as cell size, protein content and the concentrations of atrial natriuretic peptide (ANP) and B-type natriuretic peptide (BNP) were analysed for the confirmation of hypertrophy induction. Angiotensin II (100 nM) caused an increase in cell volume (69·26 (SD 1·21)%),protein content (48·48 (SD 1·64)%), ANP (81·90 (SD 1·22)%) and BNP (108·57 (SD 1·47)%). BDE treatment significantly reduced cell volume, protein content and the concentrations of ANP and BNP (P#0·05) in H9c2 cells. The activity of various antioxidant enzymes and the concentration of reduced glutathione, which was lowered due to hypertrophy, were increased in BDE-treated cells. The BDE treatment also reduced intracellular reactive oxygen species generation, lipid peroxidation and protein carbonyls in cells. In addition,the expression patterns of NF-kb and transforming growth factor b1 were found to be increased during hypertrophy, and their expressions were reduced on BDE treatment. In vitro chemical assays showed that BDE inhibits angiotensin-converting enzyme and xanthine oxidase in a dose-dependent manner with an estimated 50% effective concentration (EC50) value of 166·12 (SD 2·42) and 60·05 (SD 1·54) mg/ml,respectively. The overall results clearly indicate the therapeutic potential of B. diffusa against cardiac hypertrophy, in addition to its nutritional qualities.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Angiotensin II / metabolism*
  • Animals
  • Antioxidants / metabolism
  • Antioxidants / pharmacology
  • Biomarkers / metabolism
  • Cardiovascular Agents / pharmacology
  • Cell Line
  • Down-Regulation
  • Glutathione / metabolism
  • Hypertrophy
  • Lipid Peroxidation
  • Myoblasts, Cardiac / drug effects*
  • Myoblasts, Cardiac / metabolism
  • Myoblasts, Cardiac / pathology
  • Myocardium / metabolism
  • Myocardium / pathology*
  • NF-kappa B / metabolism*
  • Nyctaginaceae*
  • Oxidative Stress / drug effects*
  • Plant Extracts / pharmacology
  • Protein Carbonylation
  • Rats
  • Reactive Oxygen Species / metabolism
  • Transforming Growth Factor beta1 / metabolism*

Substances

  • Antioxidants
  • Biomarkers
  • Cardiovascular Agents
  • NF-kappa B
  • Plant Extracts
  • Reactive Oxygen Species
  • Transforming Growth Factor beta1
  • Angiotensin II
  • Glutathione