Abstract
Activation of the transcription factor PPARγ by the n-3 fatty acid docosahexaenoic acid (DHA) is implicated in controlling proinflammatory cytokine secretion, but the intracellular signaling pathways engaged by PPARγ are incompletely characterized. Here, we identify the adapter-encoding gene SOCS3 as a critical transcriptional target of PPARγ. SOCS3 promoter binding and gene transactivation by PPARγ was associated with a repression in differentiation of proinflammatory T-helper (TH)17 cells. Accordingly, TH17 cells induced in vitro displayed increased SOCS3 expression and diminished capacity to produce interleukin (IL)-17 following activation of PPARγ by DHA. Furthermore, naïve CD4 T cells derived from mice fed a DHA-enriched diet displayed less capability to differentiate into TH17 cells. In two different mouse models of cancer, DHA prevented tumor outgrowth and angiogenesis in an IL-17-dependent manner. Altogether, our results uncover a novel molecular pathway by which PPARγ-induced SOCS3 expression prevents IL-17-mediated cancer growth.
©2013 AACR.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Blotting, Western
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CD4-Positive T-Lymphocytes / drug effects
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CD4-Positive T-Lymphocytes / metabolism
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Cell Differentiation / drug effects
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Cell Line, Tumor
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Diet
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Docosahexaenoic Acids / administration & dosage
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Docosahexaenoic Acids / pharmacology
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Female
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Interleukin-17 / metabolism
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Mammary Neoplasms, Experimental / genetics*
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Mammary Neoplasms, Experimental / pathology
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Mammary Neoplasms, Experimental / prevention & control
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Mice
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Mice, Inbred BALB C
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Mice, Inbred C57BL
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Mice, Knockout
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Mice, Nude
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PPAR gamma / agonists
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PPAR gamma / genetics*
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PPAR gamma / metabolism
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Promoter Regions, Genetic / genetics
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RNA Interference
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Reverse Transcriptase Polymerase Chain Reaction
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Suppressor of Cytokine Signaling 3 Protein
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Suppressor of Cytokine Signaling Proteins / genetics*
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Suppressor of Cytokine Signaling Proteins / metabolism
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Th17 Cells / drug effects
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Th17 Cells / metabolism
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Transcriptional Activation*
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Tumor Burden / drug effects
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Tumor Burden / genetics
Substances
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Interleukin-17
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PPAR gamma
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Socs3 protein, mouse
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Suppressor of Cytokine Signaling 3 Protein
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Suppressor of Cytokine Signaling Proteins
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Docosahexaenoic Acids