MiR-424 regulates monocytic differentiation of human leukemia U937 cells by directly targeting CDX2

Biotechnol Lett. 2013 Nov;35(11):1799-806. doi: 10.1007/s10529-013-1264-9. Epub 2013 Jun 26.

Abstract

MiR-424 plays an important role via promoting the monocytic differentiation in many human leukemia cell lines. Here, we report that miR-424 decreased miR-125b expression to 36 % by directly targeting caudal type homeobox 2. However, miR-424 also decreased expression of Fes, PU.1 and colony-stimulating factor receptor (MCSFR). As Fes, PU.1 and MCSFR were down-regulated by over-expression of miR-125b (unpublished work), a similar effect of miR-424 and Fes siRNA on CD64, Egr-1, Egr-2 and CEBPA indicates that Fes may be an important downstream target of miR-424. We hypothesize that miR-424 promotes monocytic differentiation by regulating other critical factors and miR-424 has high affinity for these factors. For the first time, the molecular mechanism of miR-424 during monocytic differentiation of U937 cells has been elucidated in this study.

MeSH terms

  • CDX2 Transcription Factor
  • Cell Differentiation / drug effects*
  • Down-Regulation
  • Gene Expression / drug effects
  • Homeodomain Proteins / metabolism*
  • Humans
  • MicroRNAs / biosynthesis
  • MicroRNAs / metabolism*
  • Monocytes / drug effects*
  • Monocytes / physiology
  • Proto-Oncogene Proteins / biosynthesis
  • Proto-Oncogene Proteins c-fes / biosynthesis
  • Receptors, Colony-Stimulating Factor / biosynthesis
  • Trans-Activators / biosynthesis
  • U937 Cells

Substances

  • CDX2 Transcription Factor
  • CDX2 protein, human
  • Homeodomain Proteins
  • MIRN125 microRNA, human
  • MIRN424 microrna, human
  • MicroRNAs
  • Proto-Oncogene Proteins
  • Receptors, Colony-Stimulating Factor
  • Trans-Activators
  • proto-oncogene protein Spi-1
  • FES protein, human
  • Proto-Oncogene Proteins c-fes