The miRNA and mRNA Signatures of Peripheral Blood Cells in Humans Infected with Trypanosoma brucei gambiense

PLoS One. 2013 Jun 27;8(6):e67312. doi: 10.1371/journal.pone.0067312. Print 2013.

Abstract

Simple, reliable tools for diagnosis of human African Trypanosomiases could ease field surveillance and enhance patient care. In particular, current methods to distinguish patients with (stage II) and without (stage I) brain involvement require samples of cerebrospinal fluid. We describe here an exploratory study to find out whether miRNAs from peripheral blood leukocytes might be useful in diagnosis of human trypanosomiasis, or for determining the stage of the disease. Using microarrays, we measured miRNAs in samples from Trypanosoma brucei gambiense-infected patients (9 stage I, 10 stage II), 8 seronegative parasite-negative controls and 12 seropositive, but parasite-negative subjects. 8 miRNAs (out of 1205 tested) showed significantly lower expression in patients than in seronegative, parasite-negative controls, and 1 showed increased expression. There were no clear differences in miRNAs between patients in different disease stages. The miRNA profiles could not distinguish seropositive, but parasitologically negative samples from controls and results within this group did not correlate with those from the trypanolysis test. Some of the regulated miRNAs, or their predicted mRNA targets, were previously reported changed during other infectious diseases or cancer. We conclude that the changes in miRNA profiles of peripheral blood lymphocytes in human African trypanosomiasis are related to immune activation or inflammation, are probably disease-non-specific, and cannot be used to determine the disease stage. The approach has little promise for diagnostics but might yield information about disease pathology.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Biomarkers / blood
  • Gene Expression Profiling
  • Humans
  • Leukocytes / metabolism
  • MicroRNAs / blood*
  • Microarray Analysis
  • Models, Molecular
  • RNA, Messenger / blood*
  • Real-Time Polymerase Chain Reaction
  • Transcriptome
  • Trypanosoma brucei gambiense*
  • Trypanosomiasis, African / blood*

Substances

  • Biomarkers
  • MicroRNAs
  • RNA, Messenger

Grants and funding

SL was supported by a grant from the German Academic Exchange Service (Deutscher Akademischer Austauschdienst, DAAD). GS was supported by the Alexander von Humboldt Foundation. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.