Abstract
HLA-B*57 is strongly associated with immune control of HIV and delayed AIDS progression. The closely related, but less protective, HLA-B*58:01 presents similar epitopes, but HLA-B*58:01(+) individuals do not generate CD8(+) T cells targeting the KF11-Gag epitope, which has been linked to low viremia. Here we show that HLA-B*58:01 binds and presents KF11 peptide, but HIV-infected HLA-B*58:01(+) cells fail to process KF11. This unexpected finding demonstrates that immunodominance patterns can be influenced by intracellular events independent of HLA binding motifs.
Publication types
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Comparative Study
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
MeSH terms
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CD8-Positive T-Lymphocytes / immunology*
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CD8-Positive T-Lymphocytes / metabolism
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Epitopes / immunology*
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HIV Infections / immunology*
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HIV Infections / metabolism
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HIV Infections / virology
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HIV-1 / immunology*
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HLA-B Antigens / immunology*
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HLA-B Antigens / metabolism
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Humans
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Peptide Fragments / immunology
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Peptide Fragments / metabolism
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T-Lymphocytes, Cytotoxic / immunology
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T-Lymphocytes, Cytotoxic / metabolism
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Viral Load
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Viremia / immunology*
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Viremia / metabolism
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gag Gene Products, Human Immunodeficiency Virus / immunology
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gag Gene Products, Human Immunodeficiency Virus / metabolism
Substances
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Epitopes
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HLA-B Antigens
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HLA-B*58:01 antigen
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HLA-B57 antigen
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Peptide Fragments
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gag Gene Products, Human Immunodeficiency Virus