Activation of Wnt/β-catenin signaling increases apoptosis in melanoma cells treated with trail

PLoS One. 2013 Jul 15;8(7):e69593. doi: 10.1371/journal.pone.0069593. Print 2013.

Abstract

While the TRAIL pathway represents a promising therapeutic target in melanoma, resistance to TRAIL-mediated apoptosis remains a barrier to its successful adoption. Since the Wnt/β-catenin pathway has been implicated in facilitating melanoma cell apoptosis, we investigated the effect of Wnt/β-catenin signaling on regulating the responses of melanoma cells to TRAIL. Co-treatment of melanoma cell lines with WNT3A-conditioned media and recombinant TRAIL significantly enhanced apoptosis compared to treatment with TRAIL alone. This apoptosis correlates with increased abundance of the pro-apoptotic proteins BCL2L11 and BBC3, and with decreased abundance of the anti-apoptotic regulator Mcl1. We then confirmed the involvement of the Wnt/β-catenin signaling pathway by demonstrating that siRNA-mediated knockdown of an intracellular β-catenin antagonist, AXIN1, or treating cells with an inhibitor of GSK-3 also enhanced melanoma cell sensitivity to TRAIL. These studies describe a novel regulation of TRAIL sensitivity in melanoma by Wnt/β-catenin signaling, and suggest that strategies to enhance Wnt/β-catenin signaling in combination with TRAIL agonists warrant further investigation.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis Regulatory Proteins / metabolism
  • Apoptosis*
  • Axin Protein / antagonists & inhibitors
  • Bcl-2-Like Protein 11
  • Cell Culture Techniques
  • Cell Line, Tumor
  • Culture Media, Conditioned
  • Glycogen Synthase Kinase 3 / antagonists & inhibitors
  • Humans
  • Melanoma / metabolism*
  • Melanoma / pathology
  • Membrane Proteins / metabolism
  • Myeloid Cell Leukemia Sequence 1 Protein / metabolism
  • Protein Kinase Inhibitors / pharmacology
  • Proto-Oncogene Proteins / metabolism
  • RNA Interference
  • TNF-Related Apoptosis-Inducing Ligand / genetics
  • TNF-Related Apoptosis-Inducing Ligand / metabolism
  • TNF-Related Apoptosis-Inducing Ligand / pharmacology*
  • Wnt Signaling Pathway*
  • Wnt3A Protein / pharmacology*
  • beta Catenin / metabolism*

Substances

  • AXIN1 protein, human
  • Apoptosis Regulatory Proteins
  • Axin Protein
  • BBC3 protein, human
  • BCL2L11 protein, human
  • Bcl-2-Like Protein 11
  • Culture Media, Conditioned
  • MCL1 protein, human
  • Membrane Proteins
  • Myeloid Cell Leukemia Sequence 1 Protein
  • Protein Kinase Inhibitors
  • Proto-Oncogene Proteins
  • TNF-Related Apoptosis-Inducing Ligand
  • TNFSF10 protein, human
  • WNT3A protein, human
  • Wnt3A Protein
  • beta Catenin
  • Glycogen Synthase Kinase 3