High-risk human papillomavirus E6 inhibits monocyte differentiation to Langerhans cells

Virology. 2013 Sep;444(1-2):257-62. doi: 10.1016/j.virol.2013.06.020. Epub 2013 Jul 18.

Abstract

High-risk human papillomaviruses (HPVs) cause a variety of malignancies of the mucosal epithelium. However, the local immune evasion strategies used by HPV-transformed cells remain unclear. Here, we examined the effect of HPV-positive cancer cells on human peripheral blood monocytes, which are precursors of Langerhans cells, key antigen-presenting cells in the squamous epithelium. HPV-positive cervical cancer cells and HPV-E6 expressing cells inhibited monocyte differentiation to Langerhans cells in a contact-dependent manner. Unlike Langerhans cells, monocytes that differentiated in the presence of HPV16 E6-expressing cells exhibited high levels of endocytic activity. Our results suggest that cells infected by high-risk HPV evade immune surveillance by blocking the differentiation of monocytes into competent antigen presenting cells.

Keywords: Cervical cancer; Dendritic cell; Immune evasion; Monocytes.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Differentiation*
  • Cells, Cultured
  • Coculture Techniques
  • Female
  • Humans
  • Immune Evasion
  • Langerhans Cells / physiology*
  • Langerhans Cells / virology*
  • Monocytes / physiology*
  • Monocytes / virology*
  • Oncogene Proteins, Viral / metabolism
  • Papillomaviridae / immunology*
  • Papillomaviridae / pathogenicity

Substances

  • Oncogene Proteins, Viral