Evidence for triclosan-induced activation of human and rodent xenobiotic nuclear receptors

Toxicol In Vitro. 2013 Oct;27(7):2049-60. doi: 10.1016/j.tiv.2013.07.008. Epub 2013 Jul 27.

Abstract

The bacteriostat triclosan (2,4,4'-trichloro-2'-hydroxydiphenylether) (TCS) decreases rat serum thyroxine via putative nuclear receptor (NR) interaction(s) and subsequent transcriptional up-regulation of hepatic catabolism and clearance. However, due to the evolutionary divergence of the constitutive androstane and pregnane-X receptors (CAR, PXR), TCS-mediated downstream effects may be species-dependent. To test the hypothesis that TCS activates xenobiotic NRs across species, cell-based NR reporter assays were employed to assess potential activation of rat, mouse, and human PXR, and rat, mouse, and three splice variants of human CAR. TCS activated hPXR, acted as an inverse agonist of hCAR1, and as a weak agonist of hCAR3. TCS failed to activate rPXR in full-length receptor reporter assays, and instead acted as a modest inverse agonist of rCAR. Consistent with the rat data, TCS also failed to activate mPXR and was a modest inverse agonist of mCAR. These data suggest that TCS may interact with multiple NRs, including hPXR, hCAR1, hCAR3, and rCAR in order to potentially affect hepatic catabolism. Overall these data support the conclusion that TCS may interact with NRs to regulate hepatic catabolism and downstream thyroid hormone homeostasis in both rat and human models, though perhaps by divergent mechanisms.

Keywords: 1,4-bis[2-(3,5-dichloropyridyloxy)]benzene; 6-(4-chlorophenyl)imidazo[2,1-b][1,3]thiazole-5-carbaldehyde-O-(3,4-dichlorobenzyl) oxime; AOP; CITCO; CTZ; CYP; Constitutive androstane receptor; DEHP; DPX2; EC50; Hepatic catabolism; LBD; MIE; PB; Pregnane-X receptor; RPXR; T4; TCPOBOP; TCS; Thyroid disruption; Triclosan; adverse outcome pathway; cDNA; clotrimazole; complementary DNA; cytochrome P450; di-(2-ethylhexyl)phthalate; hCAR; hPXR; half-maximal effect concentration; human PXR; human constitutive androstane receptor; ligand binding domain; mCAR; mPXR; molecular-initiating event; mouse PXR; mouse constitutive androstane receptor; name of the hPXR model, commercial product of Puracyp, Inc; name of the rPXR model, commercial product of Puracyp, Inc; phenobarbital; rCAR; rPXR; rat PXR; rat constitutive androstane receptor; thyroxine; triclosan.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Animals
  • Anti-Bacterial Agents / pharmacology*
  • Anti-Infective Agents, Local / pharmacology
  • Cell Nucleus / drug effects*
  • Cell Nucleus / metabolism
  • Constitutive Androstane Receptor
  • Drug Inverse Agonism
  • Genes, Reporter / drug effects
  • HEK293 Cells
  • Hep G2 Cells
  • Hepatocytes / drug effects*
  • Hepatocytes / metabolism
  • Humans
  • Kinetics
  • Mice
  • Models, Biological*
  • Pregnane X Receptor
  • Protein Isoforms / agonists
  • Protein Isoforms / genetics
  • Protein Isoforms / metabolism
  • Rats
  • Receptors, Cytoplasmic and Nuclear / agonists*
  • Receptors, Cytoplasmic and Nuclear / genetics
  • Receptors, Cytoplasmic and Nuclear / metabolism
  • Receptors, Steroid / agonists*
  • Receptors, Steroid / genetics
  • Receptors, Steroid / metabolism
  • Recombinant Fusion Proteins / biosynthesis
  • Recombinant Fusion Proteins / chemistry
  • Recombinant Fusion Proteins / metabolism
  • Species Specificity
  • Triclosan / pharmacology*

Substances

  • Anti-Bacterial Agents
  • Anti-Infective Agents, Local
  • Constitutive Androstane Receptor
  • Pregnane X Receptor
  • Protein Isoforms
  • Receptors, Cytoplasmic and Nuclear
  • Receptors, Steroid
  • Recombinant Fusion Proteins
  • Triclosan