Confirmation of oxidative stress and fatty acid disturbances in two further Papillon-Lefèvre syndrome families with identification of a new mutation

J Eur Acad Dermatol Venereol. 2014 Aug;28(8):1049-56. doi: 10.1111/jdv.12265. Epub 2013 Sep 3.

Abstract

Background: We have previously reported oxidative and fatty acids disturbances in one Papillon-Lefèvre syndrome (PLS) family. This Mendelian condition characterized by palmar plantar keratosis and severe aggressive periodontitis, is caused by mutations in the cathepsin C (CTSC) gene. In this study, we have analysed two further unrelated PLS families to confirm this association.

Methods: Mutations were identified by direct sequencing of CTSC. Biochemical analyses were performed in probands and their relatives in order to determine plasma levels of vitamin E, CoQ10 , lipid hydroperoxides (HP) and fatty acid patterns.

Results: Pathogenic CTSC mutations were identified in both families including a new mutation (c504C>G). Both probands showed low levels of vitamin E and CoQ10 , and high levels of lipid HP, and also very low levels of docohexaenoic acid.

Conclusions: The previously reported oxidative and fatty acids disturbances were confirmed as a feature of this condition in two further families. There are low levels of antioxidant markers and high levels of oxidative markers, in addition of low levels of some anti-inflammatory fatty acids in persons suffering PLS and some of their relatives.

MeSH terms

  • Adult
  • Aged
  • Child, Preschool
  • Fatty Acids / metabolism*
  • Female
  • Humans
  • Infant
  • Male
  • Middle Aged
  • Mutation*
  • Oxidative Stress*
  • Papillon-Lefevre Disease / genetics
  • Papillon-Lefevre Disease / metabolism*
  • Pedigree
  • Polymerase Chain Reaction

Substances

  • Fatty Acids