TCF-1 controls ILC2 and NKp46+RORγt+ innate lymphocyte differentiation and protection in intestinal inflammation

J Immunol. 2013 Oct 15;191(8):4383-91. doi: 10.4049/jimmunol.1301228. Epub 2013 Sep 13.

Abstract

Innate lymphocyte populations play a central role in conferring protective immunity at the mucosal frontier. In this study, we demonstrate that T cell factor 1 (TCF-1; encoded by Tcf7), a transcription factor also important for NK and T cell differentiation, is expressed by multiple innate lymphoid cell (ILC) subsets, including GATA3(+) nuocytes (ILC2) and NKp46(+) ILCs (ILC3), which confer protection against lung and intestinal inflammation. TCF-1 was intrinsically required for the differentiation of both ILC2 and NKp46(+) ILC3. Loss of TCF-1 expression impaired the capacity of these ILC subsets to produce IL-5, IL-13, and IL-22 and resulted in crippled responses to intestinal infection with Citrobacter rodentium. Furthermore, a reduction in T-bet expression required for Notch-2-dependent development of NKp46(+) ILC3 showed a dose-dependent reduction in TCF-1 expression. Collectively, our findings demonstrate an essential requirement for TCF-1 in ILC2 differentiation and reveal a link among Tcf7, Notch, and Tbx21 in NKp46(+) ILC3 development.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antigens, Ly / metabolism
  • Cell Differentiation / immunology
  • Citrobacter rodentium / immunology
  • Enterobacteriaceae Infections / immunology
  • GATA3 Transcription Factor / metabolism
  • Hepatocyte Nuclear Factor 1-alpha
  • Inflammation / immunology
  • Inflammation / microbiology
  • Interleukin-13 / biosynthesis
  • Interleukin-22
  • Interleukin-5 / biosynthesis
  • Interleukins / biosynthesis
  • Intestines / immunology*
  • Intestines / microbiology
  • Killer Cells, Natural / metabolism*
  • Lymphocyte Activation
  • Mice
  • Mice, Knockout
  • Mucous Membrane / cytology
  • Mucous Membrane / immunology
  • Natural Cytotoxicity Triggering Receptor 1 / metabolism
  • Nuclear Receptor Subfamily 1, Group F, Member 3 / metabolism
  • Receptor, Notch2 / metabolism
  • T Cell Transcription Factor 1 / genetics
  • T Cell Transcription Factor 1 / metabolism*
  • T-Box Domain Proteins / biosynthesis
  • T-Box Domain Proteins / immunology
  • T-Lymphocytes / metabolism*

Substances

  • Antigens, Ly
  • GATA3 Transcription Factor
  • Gata3 protein, mouse
  • Hepatocyte Nuclear Factor 1-alpha
  • Hnf1a protein, mouse
  • Interleukin-13
  • Interleukin-5
  • Interleukins
  • Natural Cytotoxicity Triggering Receptor 1
  • Ncr1 protein, mouse
  • Notch2 protein, mouse
  • Nuclear Receptor Subfamily 1, Group F, Member 3
  • Receptor, Notch2
  • T Cell Transcription Factor 1
  • T-Box Domain Proteins
  • T-box transcription factor TBX21