OK-432 synergizes with IFN-γ to confer dendritic cells with enhanced antitumor immunity

Immunol Cell Biol. 2014 Mar;92(3):263-74. doi: 10.1038/icb.2013.87. Epub 2013 Dec 3.

Abstract

Generation of functional dendritic cells (DCs) with boosted immunity after the withdrawal of initial activation/maturation conditions remains a significant challenge. In this study, we investigated the impact of a newly developed maturation cocktail consisting of OK-432 and interferon-gamma (IFN-γ) on the function of human monocyte-derived DCs (MoDCs). We found that OK-432 plus IFN-γ stimulation could induce significantly stronger expression of surface molecules, production of cytokines, as well as migration of DCs compared with OK-432 stimulation alone. Most importantly, DCs matured with OK-432 plus IFN-γ-induced maintained secretion of interleukin-12 (IL-12)p70 in secondary culture after stimulus withdrawal. Functionally, OK-432 plus IFN-γ-conditioned DCs induce remarkable Th1 and Tc1 responses more effectively than OK-432 alone, even more than the use of α-type-1 cytokine cocktail. As a result, DCs matured with OK-432 plus IFN-γ can prime stronger cytotoxic lymphocyte (CTL) and natural killer (NK) cell response against tumor cells in vitro. Peripheral blood mononuclear cells activated by DCs matured with OK-432 plus IFN-γ also showed greater tumor growth inhibition in vivo in null mice. Molecular mechanistic analysis showed that DC maturation using IFN-γ in concert with OK-432 involves the activation of p38 and nuclear factor-kappa B (NF-κB) pathways. This study provided a novel strategy to generate more potent immune segments in DC vaccine.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antineoplastic Agents / pharmacology*
  • Biomarkers / metabolism
  • Cell Differentiation / drug effects
  • Cell Movement / drug effects
  • Cell Movement / immunology
  • Culture Media, Serum-Free
  • Cytokines / biosynthesis
  • Dendritic Cells / drug effects*
  • Dendritic Cells / enzymology
  • Dendritic Cells / immunology*
  • Drug Synergism
  • Female
  • Humans
  • Inflammation Mediators / metabolism
  • Interferon-gamma / pharmacology*
  • Mice
  • Mice, Inbred BALB C
  • Mice, Nude
  • Monocytes / pathology
  • NF-kappa B / metabolism
  • Neoplasms / immunology*
  • Picibanil / pharmacology*
  • Signal Transduction / drug effects
  • T-Lymphocytes, Cytotoxic / drug effects
  • T-Lymphocytes, Cytotoxic / immunology
  • Th1 Cells / drug effects
  • Th1 Cells / metabolism
  • p38 Mitogen-Activated Protein Kinases / metabolism

Substances

  • Antineoplastic Agents
  • Biomarkers
  • Culture Media, Serum-Free
  • Cytokines
  • Inflammation Mediators
  • NF-kappa B
  • Picibanil
  • Interferon-gamma
  • p38 Mitogen-Activated Protein Kinases