The effect of iron and zinc supplementation and its discontinuation on liver antioxidant status in rats fed deficient diets

Eur J Nutr. 2014 Jun;53(4):1083-92. doi: 10.1007/s00394-013-0610-y. Epub 2013 Dec 18.

Abstract

Purpose: The aim was to investigate the effect of iron or combined iron/zinc supplementation on rat liver antioxidant status.

Methods: The 6-week male Wistar rats were examined in 3 stages: (1) 4-week adaptation to the diets (C-control AIN-93M diet, D-iron deficient and R-with 50% reduction in all vitamin and mineral amounts); (2) 4-week supplementation with the same regimen enriched with tenfold more iron or iron/zinc; (3) 2-week post-supplementation period (the same diets as in the stage I).

Results: Combined iron/zinc supplementation similarly to iron supplementation alone significantly (p values ≤ 0.05) increased the iron content in the liver in D and R rats after stages II and III. Moreover, iron/zinc supplementation compared to iron supplementation alone significantly decreased the liver concentration of 8-isoprostane (after stage II in D and after stage III in R rats), protein carbonyl groups (only after stage III in R rats) and 8-hydroxy-2-deoxyguanosine (after stage II in R and after stage III in D and R rats). In rats fed R-type of diets after stage II hepatic superoxide dismutase (SOD) and catalase (CAT) activity, but not glutathione peroxidation activity and total antioxidant capacity, was lower in iron and iron/zinc supplemented than in non-supplemented rats, whereas after stage III in iron/zinc supplemented SOD was lower and CAT activity was higher in comparison with non-supplemented and iron supplemented rats.

Conclusions: The simultaneous iron/zinc supplementation can protect liver against peroxidative damage induced by high doses of iron during and after the intervention in rats fed iron-deficient diet and diet with reduced amounts of vitamins and minerals. The post-intervention observation is relevant because the effect may be delayed and visible only after this period.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 8-Hydroxy-2'-Deoxyguanosine
  • Animals
  • Antioxidants / metabolism
  • Catalase / metabolism
  • DNA Damage / drug effects
  • Deoxyguanosine / analogs & derivatives
  • Deoxyguanosine / metabolism
  • Dietary Supplements*
  • Dinoprost / analogs & derivatives
  • Dinoprost / metabolism
  • Glutathione / metabolism
  • Iron, Dietary / administration & dosage*
  • Liver / drug effects
  • Liver / metabolism*
  • Male
  • Micronutrients / administration & dosage
  • Micronutrients / deficiency*
  • Organ Size / drug effects
  • Rats
  • Rats, Wistar
  • Superoxide Dismutase / metabolism
  • Zinc / administration & dosage*

Substances

  • Antioxidants
  • Iron, Dietary
  • Micronutrients
  • 8-epi-prostaglandin F2alpha
  • 8-Hydroxy-2'-Deoxyguanosine
  • Dinoprost
  • Catalase
  • Superoxide Dismutase
  • Deoxyguanosine
  • Glutathione
  • Zinc