Bombesin-like peptides elevate cytosolic calcium in small cell lung cancer cells

Biochem Biophys Res Commun. 1987 Aug 31;147(1):189-95. doi: 10.1016/s0006-291x(87)80105-5.

Abstract

The ability of bombesin-like peptides to elevate intracellular Ca2+ levels in small cell lung cancer cells was investigated using the fluorescent Ca2+ indicator Fura 2. Nanomolar concentrations of bombesin elevated cytosolic Ca2+ levels in the absence or presence of extracellular Ca2+. Potent bombesin receptor agonists, such as gastrin releasing peptide (GRP) or (GRP)14-27 elevated cytosolic Ca2+ levels whereas inactive compounds such as (D-Trp8)bombesin or (GRP)1-16 did not. Furthermore, the bombesin receptor antagonist (D-Arg1, D-Pro2, D-Trp7,9, Leu11) substance P (30 microM) had no effect on the Ca2+ levels by itself but antagonized the increase in Ca2+ caused by 10 nM or 100 nM bombesin. These data suggest that bombesin receptors may regulate the release of Ca2+ from intracellular organelles in small cell lung cancer cells.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Benzofurans
  • Bombesin / pharmacology*
  • Calcium / metabolism*
  • Carcinoma, Small Cell / physiopathology*
  • Cell Line
  • Cytosol / metabolism
  • Fura-2
  • Gastrin-Releasing Peptide
  • Humans
  • In Vitro Techniques
  • Oligopeptides / pharmacology
  • Peptides / pharmacology
  • Receptors, Bombesin
  • Receptors, Neurotransmitter / physiology*
  • Somatostatin / pharmacology
  • Structure-Activity Relationship
  • Substance P / antagonists & inhibitors
  • Vasoactive Intestinal Peptide / pharmacology

Substances

  • Benzofurans
  • Oligopeptides
  • Peptides
  • Receptors, Bombesin
  • Receptors, Neurotransmitter
  • Substance P
  • Vasoactive Intestinal Peptide
  • Somatostatin
  • Gastrin-Releasing Peptide
  • Bombesin
  • Calcium
  • Fura-2