MiR-98 is involved in rat embryo implantation by targeting Bcl-xl

FEBS Lett. 2014 Feb 14;588(4):574-83. doi: 10.1016/j.febslet.2013.12.026. Epub 2014 Jan 18.

Abstract

In a previous study, via microRNA microarray analysis we found that miR-98 is differentially expressed in rat uteri during the peri-implantation period (unpublished data). However, the role of miR-98 in rat embryo implantation remains elusive. Here, we found that the level of miR-98 is lower on day 5 and 6 of gestation (g.d. 5-6) than that on g.d. 3-4 and g.d. 7-8 in rat. MiR-98 expression is significantly decreased by delayed implantation. Down-regulation of miR-98 promotes ESC proliferation and inhibits apoptosis. Up-regulation of miR-98 displays opposite effects. Further investigation revealed that miR-98 can bind to the 3'-untranslated region (3'-UTR) of B-cell lymphoma-extra large (Bcl-xl) to inhibit Bcl-xl translation. Collectively, down-regulation of miR-98 in rat uterus during the receptive phase is linked to the increase of cell proliferation via targeting Bcl-xl.

Keywords: Bcl-xl; Embryo implantation; Rat; Uterus; miR-98.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis / drug effects
  • Apoptosis / genetics
  • Base Sequence
  • Cell Proliferation / drug effects
  • Embryo Implantation / drug effects
  • Embryo Implantation / genetics*
  • Female
  • Gene Expression Regulation, Developmental / drug effects
  • Gene Expression Regulation, Developmental / genetics
  • Humans
  • MicroRNAs / genetics*
  • Pregnancy
  • Progesterone / pharmacology
  • Rats
  • Rats, Sprague-Dawley
  • Uterus / cytology
  • Uterus / drug effects
  • Uterus / embryology
  • Uterus / metabolism
  • bcl-X Protein / genetics*

Substances

  • MIRN98 microRNA-98, rat
  • MicroRNAs
  • bcl-X Protein
  • Progesterone