3'-Deoxy-3'-[18F]fluorothymidine positron emission tomography imaging of thymidine kinase 1 activity after 5-fluorouracil treatment in a mouse tumor model

Anticancer Res. 2014 Feb;34(2):759-66.

Abstract

Aim: We aimed to investigate whether 3'-deoxy-3'-[(18)F]fluorothymidine ([(18)F]FLT) positron emission tomography (PET) can estimate thymidine kinase 1 (TK1) activity after thymidylate synthase (TS) inhibition by 5-fluorouracil (5-FU) in a mouse tumor model.

Materials and methods: Nude mice with HT29 tumors were injected with phosphate-buffered saline or 5-FU (16.7 or 50 mg/kg). Twenty-four hours later, 2-hour dynamic images were acquired after injection of [(18)F]FLT. In another group of mice with HT29 cells, static PET images were obtained 110 min after [(18)F]FLT injection.

Results: Kinetic parameters related to [(18)F]FLT retention increased significantly, whereas the de-phosphorylation of [(18)F]FLT monophosphate decreased significantly. The standardized uptake value (SUVmean) of HT29 tumors correlated significantly with the net influx constant and the distribution volume for phosphorylated [(18)F]FLT. There was a significant correlation between the tumor SUVmean and TK1 activity.

Conclusion: SUVmean at 110-120 min after [(18)F]FLT, can quantitatively evaluate kinetic parameters and TK1 activity after TS inhibition.

Keywords: 3’-deoxy-3’-[18F]fluorothymidine; 5-fluorouracil; positron emission tomography; thymidine kinase; thymidylate synthase.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Colonic Neoplasms / diagnostic imaging*
  • Colonic Neoplasms / drug therapy
  • Colonic Neoplasms / enzymology
  • Dideoxynucleosides* / chemistry
  • Dideoxynucleosides* / pharmacokinetics
  • Disease Models, Animal
  • Fluorouracil / pharmacology*
  • HT29 Cells
  • Heterografts
  • Humans
  • Male
  • Mice
  • Mice, Inbred BALB C
  • Mice, Nude
  • Phosphorylation
  • Positron-Emission Tomography
  • Thymidine Kinase / chemistry
  • Thymidine Kinase / metabolism*
  • Thymidylate Synthase / antagonists & inhibitors
  • Thymidylate Synthase / metabolism

Substances

  • Dideoxynucleosides
  • Thymidylate Synthase
  • Thymidine Kinase
  • thymidine kinase 1
  • alovudine
  • Fluorouracil