Abstract
Parmenter and colleagues identify molecular pathways by which BRAF-MAPK signaling regulates glycolysis in melanoma, suggesting novel approaches to target these metabolic dependencies.
MeSH terms
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Glycolysis / drug effects*
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Humans
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Melanoma / genetics*
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Protein Kinase Inhibitors / pharmacology*
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Proto-Oncogene Proteins B-raf / antagonists & inhibitors*
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Proto-Oncogene Proteins B-raf / genetics*
Substances
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Protein Kinase Inhibitors
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BRAF protein, human
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Proto-Oncogene Proteins B-raf