p53 in cell invasion, podosomes, and invadopodia

Cell Adh Migr. 2014;8(3):205-14. doi: 10.4161/cam.27841.

Abstract

Cell invasion of the extracellular matrix is prerequisite to cross tissue migration of tumor cells in cancer metastasis, and vascular smooth muscle cells in atherosclerosis. The tumor suppressor p53, better known for its roles in the regulation of cell cycle and apoptosis, has ignited much interest in its function as a suppressor of cell migration and invasion. How p53 and its gain-of-function mutants regulate cell invasion remains a puzzle and a challenge for future studies. In recent years, podosomes and invadopodia have also gained center stage status as veritable apparatus specialized in cell invasion. It is not clear, however, whether p53 regulates cell invasion through podosomes and invadopodia. In this review, evidence supporting a negative role of p53 in podosomes formation in vascular smooth muscle cells will be surveyed, and signaling nodes that may mediate this regulation in other cell types will be explored.

Keywords: atherosclerosis; cancer; cell invasion; cell migration; invadopodia; p53; podosomes; smooth muscle cells.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Actin Cytoskeleton / metabolism
  • Animals
  • Cell Adhesion / physiology
  • Cell Movement / physiology
  • Extracellular Matrix / metabolism
  • Humans
  • Neoplasms / metabolism
  • Neoplasms / pathology
  • Tumor Suppressor Protein p53 / metabolism*

Substances

  • Tumor Suppressor Protein p53