Abstract
The angiopoietin-like protein 4 (angptl4, also known as peroxisome proliferator-activated receptor [PPAR]γ-induced angiopoietin-related protein) is a multifunctional protein associated with acute-phase response. The mechanisms accounting for the increase in angptl4 expression are largely unknown. This study shows that human α1-antitrypsin (A1AT) upregulates expression and release of angplt4 in human blood adherent mononuclear cells and in primary human lung microvascular endothelial cells in a concentration- and time-dependent manner. Mononuclear cells treated for 1 h with A1AT (from 0.1 to 4 mg/ml) increased mRNA of angptl4 from 2- to 174-fold, respectively, relative to controls. In endothelial cells, the maximal effect on angptl4 expression was achieved at 8 h with 2 mg/ml A1AT (11-fold induction versus controls). In 10 emphysema patients receiving A1AT therapy (Prolastin), plasma angptl4 levels were higher relative to patients without therapy (nanograms per milliliter, mean [95% confidence interval] 127.1 [99.5-154.6] versus 76.8 [54.8-98.8], respectively, p = 0.045) and correlated with A1AT levels. The effect of A1AT on angptl4 expression was significantly diminished in cells pretreated with a specific inhibitor of ERK1/2 activation (UO126), irreversible and selective PPARγ antagonist (GW9662), or genistein, a ligand for PPARγ. GW9662 did not alter the ability of A1AT to induce ERK1/2 phosphorylation, suggesting that PPARγ is a critical mediator in the A1AT-driven angptl4 expression. In contrast, the forced accumulation of HIF-1α, an upregulator of angptl4 expression, enhanced the effect of A1AT. Thus, acute-phase protein A1AT is a physiological regulator of angptl4, another acute-phase protein.
Copyright © 2014 by The American Association of Immunologists, Inc.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Angiopoietin-Like Protein 1
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Angiopoietin-like Proteins
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Angiopoietins / immunology*
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Angiopoietins / metabolism
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Anilides / pharmacology
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Emphysema / drug therapy
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Emphysema / immunology
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Emphysema / pathology
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Endothelial Cells / immunology*
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Endothelial Cells / metabolism
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Endothelial Cells / pathology
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Female
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Gene Expression Regulation / drug effects
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Gene Expression Regulation / immunology*
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Humans
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Hypoxia-Inducible Factor 1, alpha Subunit / genetics
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Hypoxia-Inducible Factor 1, alpha Subunit / immunology
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Leukocytes, Mononuclear / immunology*
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Leukocytes, Mononuclear / metabolism
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Leukocytes, Mononuclear / pathology
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MAP Kinase Signaling System / drug effects
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MAP Kinase Signaling System / immunology
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Male
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Mitogen-Activated Protein Kinase 1 / immunology
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Mitogen-Activated Protein Kinase 1 / metabolism
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Mitogen-Activated Protein Kinase 3 / immunology
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Mitogen-Activated Protein Kinase 3 / metabolism
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Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha
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Phosphorylation / drug effects
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Phosphorylation / immunology
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Serine Proteinase Inhibitors / immunology
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Serine Proteinase Inhibitors / pharmacology
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Transcription Factors / immunology
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Transcription Factors / metabolism
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Transcription, Genetic / drug effects
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Transcription, Genetic / immunology*
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alpha 1-Antitrypsin / immunology*
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alpha 1-Antitrypsin / metabolism
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alpha 1-Antitrypsin / pharmacology
Substances
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2-chloro-5-nitrobenzanilide
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ANGPTL1 protein, human
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Angiopoietin-Like Protein 1
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Angiopoietin-like Proteins
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Angiopoietins
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Anilides
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HIF1A protein, human
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Hypoxia-Inducible Factor 1, alpha Subunit
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Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha
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Ppargc1a protein, rat
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Serine Proteinase Inhibitors
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Transcription Factors
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alpha 1-Antitrypsin
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MAPK1 protein, human
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Mitogen-Activated Protein Kinase 1
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Mitogen-Activated Protein Kinase 3