miR-155 regulates the proliferation and cell cycle of colorectal carcinoma cells by targeting E2F2

Biotechnol Lett. 2014 Sep;36(9):1743-52. doi: 10.1007/s10529-014-1540-3. Epub 2014 May 4.

Abstract

MicroRNAs play important roles in carcinogenesis by negatively regulating the expression of target genes. Here we explore the biological function of miR-155 and the underlying mechanism in colorectal carcinoma. We validate, for the first time, that E2F2 is a direct target of miR-155 using western blot and a luciferase reporter assay and that miR-155 regulates the proliferation and cell cycle of colorectal carcinoma cells by targeting E2F2 using siRNA technology. We also found, for the first, time that E2F2 acts as a tumor suppressor in colorectal carcinoma. Overall, miR-155 plays an important role in colorectal carcinoma tumorigenesis by negative regulation of its targets including E2F2 and may be a potential therapeutic target for colorectal carcinoma treatment.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Blotting, Western
  • Cell Cycle*
  • Cell Proliferation*
  • Colorectal Neoplasms / pathology*
  • E2F2 Transcription Factor / genetics
  • E2F2 Transcription Factor / metabolism*
  • Gene Expression Regulation*
  • Gene Silencing
  • Genes, Reporter
  • Genes, Tumor Suppressor*
  • Luciferases / analysis
  • Luciferases / genetics
  • MicroRNAs / metabolism*

Substances

  • E2F2 Transcription Factor
  • E2F2 protein, human
  • MIRN155 microRNA, human
  • MicroRNAs
  • Luciferases