PTPN3 mutations and HBV may exert synergistic effects in the origin of the intrahepatic cholangiocarcinoma

Gastroenterology. 2014 Sep;147(3):719-20. doi: 10.1053/j.gastro.2014.05.046. Epub 2014 Jul 27.

Authors

Vincenzo Cardinale¹, Domenico Alvaro²

Affiliations

¹ Department of Medico-Surgical Sciences and Biotechnologies, Polo Pontino.
² Department of Medico-Surgical Sciences and Biotechnologies, Polo Pontino and Eleonora Lorillard Spencer-Cenci Foundation, Rome, Italy.

PMID: 25075940
DOI: 10.1053/j.gastro.2014.05.046

No abstract available

Publication types

Letter
Comment

MeSH terms

Bile Duct Neoplasms / genetics*
Bile Ducts, Intrahepatic / enzymology*
Cell Movement*
Cell Proliferation*
Cholangiocarcinoma / genetics*
Humans
Liver Neoplasms / genetics*
Mutation*
Neoplasm Recurrence, Local*
Protein Tyrosine Phosphatase, Non-Receptor Type 3 / genetics*

Substances

Protein Tyrosine Phosphatase, Non-Receptor Type 3