Environmental enrichment restores cognitive deficits induced by experimental childhood meningitis

Braz J Psychiatry. 2014 Oct-Dec;36(4):322-9. doi: 10.1590/1516-4446-2014-1443. Epub 2014 Jul 25.

Abstract

Objective: To evaluate the influence of environmental enrichment (EE) on memory, cytokines, and brain-derived neurotrophic factor (BDNF) in the brain of adult rats subjected to experimental pneumococcal meningitis during infancy.

Methods: On postnatal day 11, the animals received either artificial cerebrospinal fluid (CSF) or Streptococcus pneumoniae suspension intracisternally at 1 × 10(6) CFU/mL and remained with their mothers until age 21 days. Animals were divided into the following groups: control, control + EE, meningitis, and meningitis + EE. EE began at 21 days and continued until 60 days of age (adulthood). EE consisted of a large cage with three floors, ramps, running wheels, and objects of different shapes and textures. At 60 days, animals were randomized and subjected to habituation to the open-field task and the step-down inhibitory avoidance task. After the tasks, the hippocampus and CSF were isolated for analysis.

Results: The meningitis group showed no difference in performance between training and test sessions of the open-field task, suggesting habituation memory impairment; in the meningitis + EE group, performance was significantly different, showing preservation of habituation memory. In the step-down inhibitory avoidance task, there were no differences in behavior between training and test sessions in the meningitis group, showing aversive memory impairment; conversely, differences were observed in the meningitis + EE group, demonstrating aversive memory preservation. In the two meningitis groups, IL-4, IL-10, and BDNF levels were increased in the hippocampus, and BDNF levels in the CSF.

Conclusions: The data presented suggest that EE, a non-invasive therapy, enables recovery from memory deficits caused by neonatal meningitis.

Publication types

  • Evaluation Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Brain / physiopathology
  • Brain-Derived Neurotrophic Factor / blood*
  • Cognition Disorders / therapy*
  • Cytokines / blood*
  • Disease Models, Animal
  • Environmental Exposure*
  • Enzyme-Linked Immunosorbent Assay
  • Male
  • Memory Disorders / therapy*
  • Meningitis, Pneumococcal / therapy*
  • Neurogenesis / physiology
  • Neuronal Plasticity / physiology
  • Rats, Wistar
  • Recovery of Function
  • Reproducibility of Results
  • Treatment Outcome

Substances

  • Brain-Derived Neurotrophic Factor
  • Cytokines