Quantitative prediction of transporter- and enzyme-mediated clinical drug-drug interactions of organic anion-transporting polypeptide 1B1 substrates using a mechanistic net-effect model

J Pharmacol Exp Ther. 2014 Oct;351(1):214-23. doi: 10.1124/jpet.114.215970. Epub 2014 Aug 8.

Abstract

Quantitative prediction of complex drug-drug interactions (DDIs) involving hepatic transporters and cytochromes P450 (P450s) is challenging. We evaluated the extent of DDIs of nine victim drugs-which are substrates to organic anion-transporting polypeptide 1B1 and undergo P450 metabolism or biliary elimination-caused by five perpetrator drugs, using in vitro data and the proposed extended net-effect model. Hepatobiliary transport and metabolic clearance estimates were obtained from in vitro studies. Of the total of 62 clinical interaction combinations assessed using the net-effect model, 58 (94%) could be predicted within a 2-fold error, with few false-negative predictions. Model predictive performance improved significantly when in vitro active uptake clearance was corrected to recover in vivo clearance. The basic R-value model yielded only 63% predictions within 2-fold error. This study demonstrates that the interactions involving transporter-enzyme interplay need to be mechanistically assessed for quantitative rationalization and prospective prediction.

MeSH terms

  • Anti-Infective Agents / pharmacokinetics
  • Anti-Infective Agents / pharmacology
  • Antihypertensive Agents / pharmacokinetics
  • Antihypertensive Agents / pharmacology
  • Cells, Cultured
  • Cytochrome P-450 Enzyme System / metabolism*
  • Drug Interactions*
  • Female
  • Hepatocytes / drug effects
  • Hepatocytes / metabolism*
  • Humans
  • Hypolipidemic Agents / pharmacokinetics
  • Hypolipidemic Agents / pharmacology
  • Male
  • Models, Biological*
  • Organic Anion Transporters / metabolism*

Substances

  • Anti-Infective Agents
  • Antihypertensive Agents
  • Hypolipidemic Agents
  • Organic Anion Transporters
  • Cytochrome P-450 Enzyme System