Anti-PD-1-targeted therapies focusing on lymphatic malignancies: biological rationale, clinical challenges and opportunities

Acta Haematol. 2015;133(2):129-35. doi: 10.1159/000362151. Epub 2014 Sep 20.

Abstract

Cancer immunotherapy with tumor-directed antibodies has generally been very successful, while T-cell immunotherapy has been less effective. Some lymphoid malignancies can be cured with immunochemotherapy but nevertheless many patients relapse or progress in spite of maximal therapy. Both solid tumors and lymphoid malignancies develop mechanisms in order to escape destruction by the intact immune system. One such mechanism is mediated through immune checkpoints. PD-1 (programmed cell death protein-1, which is expressed on activated T and B cells, natural killer cells and myeloid cells, is one of those checkpoints. This review focuses on the effect of PD-1 activation on lymphoid malignancies and its role as a therapeutic target.

Publication types

  • Review

MeSH terms

  • Antibodies, Neoplasm / therapeutic use*
  • B-Lymphocytes / metabolism
  • B-Lymphocytes / pathology
  • Gene Expression Regulation, Neoplastic / drug effects
  • Hematologic Neoplasms / drug therapy*
  • Hematologic Neoplasms / metabolism
  • Hematologic Neoplasms / pathology
  • Humans
  • Neoplasm Proteins / antagonists & inhibitors*
  • Neoplasm Proteins / biosynthesis
  • Programmed Cell Death 1 Receptor / antagonists & inhibitors*
  • Programmed Cell Death 1 Receptor / biosynthesis
  • T-Lymphocytes / metabolism
  • T-Lymphocytes / pathology

Substances

  • Antibodies, Neoplasm
  • Neoplasm Proteins
  • PDCD1 protein, human
  • Programmed Cell Death 1 Receptor