Autophagy gene Atg16L1 prevents lethal T cell alloreactivity mediated by dendritic cells

Immunity. 2014 Oct 16;41(4):579-91. doi: 10.1016/j.immuni.2014.09.011. Epub 2014 Oct 9.

Abstract

Atg16L1 mediates the cellular degradative process of autophagy and is considered a critical regulator of inflammation based on its genetic association with inflammatory bowel disease. Here we find that Atg16L1 deficiency leads to an exacerbated graft-versus-host disease (GVHD) in a mouse model of allogeneic hematopoietic stem cell transplantation (allo-HSCT). Atg16L1-deficient allo-HSCT recipients with GVHD displayed increased T cell proliferation due to increased dendritic cell (DC) numbers and costimulatory molecule expression. Reduced autophagy within DCs was associated with lysosomal abnormalities and decreased amounts of A20, a negative regulator of DC activation. These results broaden the function of Atg16L1 and the autophagy pathway to include a role in limiting a DC-mediated response during inflammatory disease, such as GVHD.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Autophagy / immunology
  • Autophagy-Related Proteins
  • B7-1 Antigen / biosynthesis
  • B7-2 Antigen / biosynthesis
  • CD4-Positive T-Lymphocytes / immunology*
  • CD40 Antigens / biosynthesis
  • CD8-Positive T-Lymphocytes / immunology*
  • Carrier Proteins / genetics
  • Carrier Proteins / immunology*
  • Cell Proliferation
  • Cells, Cultured
  • Colitis / immunology
  • Cysteine Endopeptidases / biosynthesis
  • Dendritic Cells / immunology*
  • Disease Models, Animal
  • Female
  • Graft vs Host Disease / immunology*
  • Granulocyte-Macrophage Colony-Stimulating Factor / pharmacology
  • Hematopoietic Stem Cell Transplantation
  • Homeodomain Proteins / genetics
  • Immediate-Early Proteins / biosynthesis
  • Inflammation / immunology
  • Intracellular Signaling Peptides and Proteins / biosynthesis
  • Lymphocyte Activation / immunology
  • Lysosomes / pathology
  • Membrane Proteins / biosynthesis
  • Mice
  • Mice, Inbred BALB C
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Receptors, Antigen, T-Cell, gamma-delta / immunology
  • Transplantation, Homologous
  • Tumor Necrosis Factor alpha-Induced Protein 3

Substances

  • Atg16l1 protein, mouse
  • Autophagy-Related Proteins
  • B7-1 Antigen
  • B7-2 Antigen
  • CD40 Antigens
  • Carrier Proteins
  • Cd86 protein, mouse
  • Homeodomain Proteins
  • Immediate-Early Proteins
  • Intracellular Signaling Peptides and Proteins
  • Laptm5 protein, mouse
  • Membrane Proteins
  • Receptors, Antigen, T-Cell, gamma-delta
  • RAG-1 protein
  • Granulocyte-Macrophage Colony-Stimulating Factor
  • Tumor Necrosis Factor alpha-Induced Protein 3
  • Cysteine Endopeptidases
  • Tnfaip3 protein, mouse

Associated data

  • GEO/GSE61559
  • GEO/GSE61560