Human IL-21 and IL-21R deficiencies: two novel entities of primary immunodeficiency

Curr Opin Pediatr. 2014 Dec;26(6):704-12. doi: 10.1097/MOP.0000000000000160.

Abstract

Purpose of review: This review highlights the recent identification of human interleukin-21 (IL-21) and interleukin-21 receptor (IL-21R) deficiencies as novel entities of primary immunodeficiency.

Recent findings: We recently described the first patients with IL-21R deficiency who had cryptosporidial infections associated with chronic cholangitis and liver disease. All IL-21R-deficient patients suffered from recurrent respiratory tract infections. Immunological work-up revealed impaired B cell proliferation and immunoglobulin class-switch, reduced T cell effector functions, and variable natural killer cell dysfunctions. Recently, these findings have been extended by the discovery of one patient with a mutation in the IL21 gene. This patient predominantly manifested with very early onset inflammatory bowel disease and recurrent respiratory infections. Laboratory examination showed reduced circulating B cells and impaired B cell class-switch.

Summary: Human IL-21 and IL-21R deficiencies cause severe, primary immunodeficiency reminiscent of common variable immunodeficiency. Early diagnosis is critical to prevent life-threatening complications, such as secondary liver failure. In view of the critical role of IL-21 in controlling immune homeostasis, early hematopoietic stem cell transplantation might be considered as therapeutic intervention in affected children.

Publication types

  • Research Support, N.I.H., Intramural
  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Child
  • Humans
  • Immunologic Deficiency Syndromes / genetics*
  • Interleukins / deficiency*
  • Interleukins / genetics
  • Receptors, Interleukin-21 / deficiency*
  • Receptors, Interleukin-21 / genetics

Substances

  • Interleukins
  • Receptors, Interleukin-21
  • interleukin-21