Balance of Treg versus T-effector cells during systemic treatment with adalimumab and topical treatment with calcipotriol-betamethasone dipropionate ointment

Exp Dermatol. 2015 Jan;24(1):65-7. doi: 10.1111/exd.12575. Epub 2014 Nov 18.

Abstract

Diminished suppressive capacity of regulatory T cells (Treg) has been demonstrated in blood and in lesional skin of psoriatic patients. Treatment with anti-TNFα restored the number and function of circulating Treg in psoriasis. We aimed to study Treg in the skin of psoriatic patients undergoing topical treatment with calcipotriol-betamethasone dipropionate (CBD) ointment (n = 12) or systemic treatment with anti-TNFα agent adalimumab (n = 10). Skin biopsies were collected from patients with chronic plaque psoriasis who responded to the above-mentioned treatments with a SUM-score improvement of at least 50% (at the end of treatment). Biopsies were processed for immunohistochemistry. As Treg function is associated with a numerical balance between Treg and effector T cells, Foxp3/CD4 ratios were calculated. It appeared that both treatments cause a significant decrease in the presence of Foxp3+ cells. However, in patients that were treated with CBD ointment, we observed lower Foxp3/CD4 ratios after 8 weeks of treatment compared to baseline (t = 0: 0.41 ± 0.08; t = 8: 0.22 ± 0.04, P = 0.033), whereas in patients who were treated with adalimumab we observed an increase of the Foxp3/CD4 ratios after 1.5 and 16 weeks of treatment compared to baseline (t = 0: 0.25 ± 0.04; t = 1.5: 0.32 ± 0.06; t = 16: 0.49 ± 0.10, P = 0.15). Based on Foxp3/CD4 ratios, we can conclude that adalimumab treated skin differs from CBD treated skin with regard to the anti-inflammatory/inflammatory balance. We suggest that, in contrast to CBD ointment, adalimumab favours local Treg function in the skin.

Keywords: Foxp3/CD4 ratio; Treg; adalimumab; calcipotriol-betamethasone dipropionate ointment.

Publication types

  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adalimumab
  • Administration, Topical
  • Aged
  • Anti-Inflammatory Agents / chemistry
  • Antibodies, Monoclonal, Humanized / therapeutic use*
  • Betamethasone / administration & dosage
  • Betamethasone / analogs & derivatives*
  • Biopsy
  • CD4-Positive T-Lymphocytes / cytology
  • Calcitriol / administration & dosage
  • Calcitriol / analogs & derivatives*
  • Female
  • Forkhead Transcription Factors / metabolism
  • Humans
  • Immunohistochemistry
  • Inflammation / pathology
  • Male
  • Middle Aged
  • Ointments
  • Psoriasis / drug therapy*
  • Psoriasis / physiopathology
  • Skin / drug effects
  • T-Lymphocyte Subsets / cytology*
  • T-Lymphocytes, Regulatory / cytology*
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Anti-Inflammatory Agents
  • Antibodies, Monoclonal, Humanized
  • FOXP3 protein, human
  • Forkhead Transcription Factors
  • Ointments
  • Tumor Necrosis Factor-alpha
  • calcipotriene
  • betamethasone-17,21-dipropionate
  • Betamethasone
  • Calcitriol
  • Adalimumab