Increased expression of T cell immunoglobulin and mucin domain 4 is positively associated with the disease severity of patients with ankylosing spondylitis

Inflammation. 2015;38(3):935-40. doi: 10.1007/s10753-014-0055-3.

Abstract

Monocytes and associated cytokines have been shown to be involved in the pathogenesis of ankylosing spondylitis (AS). T cell immunoglobulin and mucin domain 4 (Tim-4) was identified on monocytes/macrophages and dentritic cells (DCs) and plays important roles in regulating the activities of macrophages and DCs. The current study investigated the association between Tim-4 expression and AS. Our results showed that Tim-4 expression on monocytes and Tim-4 level in plasma were highly increased in AS patients than in controls. Furthermore, TNF-α production and bath ankylosing spondylitis disease activity index (BASDAI) have positive relationships with Tim-4 expression in AS patients. High expression of Tim-4 was thought to contribute to the pathogenesis and an underlying mechanism of AS.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Autoimmune Diseases / immunology
  • Autoimmune Diseases / pathology
  • Blood Sedimentation
  • C-Reactive Protein / metabolism
  • Dendritic Cells / immunology
  • Female
  • Humans
  • Macrophages / immunology
  • Male
  • Membrane Proteins / biosynthesis
  • Membrane Proteins / blood*
  • Monocytes / metabolism*
  • Severity of Illness Index
  • Spondylitis, Ankylosing / immunology*
  • Spondylitis, Ankylosing / pathology*
  • Tumor Necrosis Factor-alpha / biosynthesis*

Substances

  • Membrane Proteins
  • TIMD4 protein, human
  • Tumor Necrosis Factor-alpha
  • C-Reactive Protein