Orf Infection in a Patient with Stat1 Gain-of-Function

J Clin Immunol. 2015 Jan;35(1):80-3. doi: 10.1007/s10875-014-0111-7. Epub 2014 Nov 4.

Abstract

Purpose: Chronic Mucocutaneous Candidiasis (CMC) refers to a group of immunodeficiencies, characterized by persistent or recurrent infections of the skin, nails, and mucosae caused by Candida. It is typically caused by inborn errors of IL-17 immunity. Orf, also known as contagious ecthyma, is a zoonotic infection caused by a dermatotropic parapoxvirus that commonly infects sheep and goats; it is transmitted to humans through contact with an infected animal or fomites. While orf is usually a benign self-limiting illness, it can be progressive and even life-threatening in immune-compromised hosts.

Methods and results: A 34-year-old man with autosomal dominant CMC due to a heterozygous STAT1 gain-of-function (GOF) mutation cut his hand with a knife during slaughter. Giant orf infection developed in 2 weeks. He was successfully treated by cidofovir injections every other week for 4 months.

Conclusions: This is the first patient with severe orf in the context of a well-defined genetically identified PID: CMC and inborn error of IL-17 immunity due to a GOF STAT1 mutation.

Keywords: Chronic mucocutaneous candidiasis; STAT1 gain-of-function; orf infection; primary immunodeficiency.

Publication types

  • Case Reports

MeSH terms

  • Adult
  • Animals
  • Antiviral Agents / therapeutic use
  • Base Sequence
  • Candidiasis, Chronic Mucocutaneous / etiology
  • Candidiasis, Chronic Mucocutaneous / genetics
  • Candidiasis, Chronic Mucocutaneous / immunology
  • Cidofovir
  • Cytosine / analogs & derivatives
  • Cytosine / therapeutic use
  • DNA Mutational Analysis
  • Ecthyma, Contagious / drug therapy
  • Ecthyma, Contagious / etiology
  • Ecthyma, Contagious / genetics*
  • Humans
  • Immunologic Deficiency Syndromes / complications
  • Immunologic Deficiency Syndromes / genetics
  • Immunologic Deficiency Syndromes / immunology
  • Interleukin-17 / metabolism
  • Male
  • Mutation, Missense*
  • Organophosphonates / therapeutic use
  • STAT1 Transcription Factor / genetics*
  • Sheep, Domestic
  • Zoonoses / genetics
  • Zoonoses / immunology

Substances

  • Antiviral Agents
  • Interleukin-17
  • Organophosphonates
  • STAT1 Transcription Factor
  • STAT1 protein, human
  • Cytosine
  • Cidofovir