Arctigenin in combination with quercetin synergistically enhances the antiproliferative effect in prostate cancer cells

Mol Nutr Food Res. 2015 Feb;59(2):250-61. doi: 10.1002/mnfr.201400558. Epub 2014 Dec 5.

Abstract

Scope: We investigated whether a combination of two promising chemopreventive agents arctigenin (Arc) and quercetin (Q) increases the anticarcinogenic potency at lower concentrations than necessary when used individually in prostate cancer.

Methods and results: Androgen-dependent LAPC-4 and LNCaP prostate cancer cells were treated with low doses of Arc and Q alone or in combination for 48 h. The antiproliferative activity of Arc was 10- to 20-fold stronger than Q in both cell lines. Their combination synergistically enhanced the antiproliferative effect, with a stronger effect in androgen receptor (AR) wild-type LAPC-4 cells than in AR mutated LNCaP cells. Arc demonstrated a strong ability to inhibit AR protein expression in LAPC-4 cells. The combination treatment significantly inhibited both AR and PI3K/Akt pathways compared to control. A protein array analysis revealed that the mixture targets multiple pathways particularly in LAPC-4 cells including Stat3 pathway. The mixture significantly inhibited the expression of several oncogenic microRNAs including miR-21, miR-19b, and miR-148a compared to control. The mixture also enhanced the inhibition of cell migration in both cell lines compared to individual compounds tested.

Conclusion: The combination of Arc and Q that target similar pathways, at low physiological doses, provides a novel regimen with enhanced chemoprevention in prostate cancer.

Keywords: Arctigenin; Combination; MicroRNA; Prostate cancer; Quercetin.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Androgens / metabolism
  • Antineoplastic Agents / pharmacology*
  • Cell Line, Tumor / drug effects
  • Cell Proliferation / drug effects*
  • Dose-Response Relationship, Drug
  • Drug Synergism
  • Furans / pharmacology*
  • Humans
  • Lignans / pharmacology*
  • Male
  • MicroRNAs / genetics
  • MicroRNAs / metabolism
  • Phosphatidylinositol 3-Kinases / genetics
  • Phosphatidylinositol 3-Kinases / metabolism
  • Prostatic Neoplasms / pathology*
  • Proto-Oncogene Proteins c-akt / genetics
  • Proto-Oncogene Proteins c-akt / metabolism
  • Quercetin / pharmacology*
  • Receptors, Androgen / genetics
  • Receptors, Androgen / metabolism
  • STAT3 Transcription Factor / genetics
  • STAT3 Transcription Factor / metabolism
  • Signal Transduction

Substances

  • AR protein, human
  • Androgens
  • Antineoplastic Agents
  • Furans
  • Lignans
  • MIRN148 microRNA, human
  • MIRN19 microRNA, human
  • MIRN21 microRNA, human
  • MicroRNAs
  • Receptors, Androgen
  • STAT3 Transcription Factor
  • STAT3 protein, human
  • Quercetin
  • Phosphatidylinositol 3-Kinases
  • Proto-Oncogene Proteins c-akt
  • arctigenin