Endothelin enhances adrenergic vasoconstriction in perfused rat mesenteric arteries

Biochem Biophys Res Commun. 1989 Mar 31;159(3):1304-8. doi: 10.1016/0006-291x(89)92252-3.

Abstract

The interaction of endothelin with alpha-adrenergic receptors was examined in isolated perfused rat mesenteric arteries. Infusion of porcine or rat endothelin increased the baseline perfusion pressure dose-dependently. Subpressor doses of both porcine (10(-11) and 10(-10)M) and rat (10(-10) and 10(-9)M) endothelin enhanced the pressor responses to norepinephrine. Nicardipine (10(-7)M), a calcium channel blocker, attenuated this potentiation. These results suggest that endothelin enhances the responsiveness of alpha-adrenergic receptors to catecholamines probably through the increase in calcium influx. Thus endothelin may interact with sympathetic nerve activity in addition to having a direct vasoconstrictor action in peripheral vascular tissue.

MeSH terms

  • Animals
  • Endothelins
  • Endothelium / physiology*
  • In Vitro Techniques
  • Male
  • Mesenteric Arteries / drug effects
  • Mesenteric Arteries / physiology*
  • Muscle, Smooth, Vascular / drug effects
  • Muscle, Smooth, Vascular / physiology*
  • Nicardipine / pharmacology
  • Norepinephrine / pharmacology*
  • Peptides / pharmacology*
  • Rats
  • Rats, Inbred Strains
  • Receptors, Adrenergic, alpha / drug effects
  • Receptors, Adrenergic, alpha / physiology*
  • Reference Values
  • Swine
  • Vasoconstriction / drug effects*

Substances

  • Endothelins
  • Peptides
  • Receptors, Adrenergic, alpha
  • Nicardipine
  • Norepinephrine