New model for the secondary structure of the 5' non-coding RNA of poliovirus is supported by biochemical and genetic data that also show that RNA secondary structure is important in neurovirulence

J Mol Biol. 1989 May 20;207(2):379-92. doi: 10.1016/0022-2836(89)90261-1.

Abstract

A secondary structure model for the 5' non-coding RNA of poliovirus has been derived by comparing computer-generated folding patterns of equivalent sequences from a number of related enteroviruses and rhinoviruses and identifying compensating mutations that suggest conservation of a common secondary structure. Although certain elements are similar, the new model differs considerably from a previously published minimal energy structure and is consistent with the observed sensitivity of in vitro RNA transcripts of infectious poliovirus cDNA to RNases and modifying chemicals. The sequence of a neurovirulent revertant of an attenuated mutant provides additional evidence for an interaction between a region known to be important for neurovirulence, sequence 471-483, and nucleotides 528 to 538.

MeSH terms

  • Animals
  • Base Sequence
  • Mice
  • Models, Genetic
  • Molecular Sequence Data
  • Mutation
  • Nucleic Acid Conformation
  • Poliovirus / genetics*
  • Poliovirus / metabolism
  • Poliovirus / pathogenicity
  • RNA, Viral / genetics*
  • RNA, Viral / metabolism
  • Spinal Cord / microbiology

Substances

  • RNA, Viral