Bone and metabolic complications of urinary diversions
Endocrinol Nutr. 2015 Feb;62(2):100-5.
doi: 10.1016/j.endonu.2014.10.007.
Epub 2014 Dec 4.
[Article in
English,
Spanish]
Affiliations
- 1 Servicio de Nefrología, Hospital Universitario Príncipe de Asturias, Alcalá de Henares, Madrid, España. Electronic address: [email protected].
- 2 Servicio de Urología, Hospital Universitario Príncipe de Asturias, Alcalá de Henares, Madrid, España.
Abstract
Hyperchloremic metabolic acidosis is a complication of urinary diversion using ileum or colon. Its prevalence ranges from 25% and 46% depending on the procedure used and renal function of the patient. It is a consequence of intestinal fluid and electrolyte exchange between intestinal mucosa and urine. The main mechanism is absorption of ammonium and chloride from urine. Long-term chronic metabolic acidosis in these patients may lead to impaired bone metabolism and osteomalacia. Regular monitoring of pH, chlorine, bicarbonate, and calcium-phosphorus metabolism is therefore essential for early diagnosis and treatment.
Keywords:
Acidosis metabólica; Conducto ileal; Derivación urinaria; Hipopotasemia; Hypokalemia; Ileal conduit; Metabolic acidosis; Neobladder; Neovejiga; Urinary diversion.
Copyright © 2014 SEEN. Published by Elsevier España, S.L.U. All rights reserved.
MeSH terms
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Acidosis / etiology*
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Alkalosis / etiology
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Bicarbonates / blood
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Bone Diseases, Metabolic / etiology*
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Calcium / blood
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Chlorides / blood
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Colon / surgery
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Gastric Mucosa / metabolism
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Humans
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Hypokalemia / etiology
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Intestinal Absorption
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Intestinal Mucosa / metabolism
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Intestine, Small / surgery
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Osteomalacia / etiology
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Phosphorus / blood
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Postoperative Complications / etiology*
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Stomach / surgery
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Urinary Diversion / adverse effects*
Substances
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Bicarbonates
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Chlorides
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Phosphorus
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Calcium