Cholecystokinin elevates cytosolic calcium in small cell lung cancer cells

Biochem Biophys Res Commun. 1989 Aug 30;163(1):605-10. doi: 10.1016/0006-291x(89)92180-3.

Abstract

The ability of cholecystokinin (CCK) to elevate intracellular Ca2+ levels in small cell lung cancer cells was investigated using the fluorescent Ca2+ indicator Fura 2. CCK-8 elevated the cytosolic Ca2+ levels in cell line NCI-H345 in a dose dependent manner. Nanomolar concentration of CCK-8 elevated cytosolic Ca2+ levels in the absence or presence of extracellular Ca2+. Potent CCK agonists such as gastrin-1 and nonsulfated CCK-8 but not inactive compounds such as CCK-27-32-NH2 elevated cytosolic Ca2+ levels. These data suggest that CCK receptors may regulate the release of Ca2+ from intracellular organelles in small cell lung cancer cells.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Bombesin / pharmacology
  • Calcium / metabolism*
  • Carcinoma, Small Cell / metabolism*
  • Cholecystokinin / pharmacology*
  • Cytosol / metabolism
  • Lung Neoplasms / metabolism*
  • Neurotensin / pharmacology
  • Receptors, Cell Surface / physiology
  • Sincalide / pharmacology
  • Structure-Activity Relationship

Substances

  • Receptors, Cell Surface
  • Neurotensin
  • Cholecystokinin
  • Sincalide
  • Bombesin
  • Calcium