Mirtoselect, an anthocyanin-rich bilberry extract, attenuates non-alcoholic steatohepatitis and associated fibrosis in ApoE(∗)3Leiden mice

J Hepatol. 2015 May;62(5):1180-6. doi: 10.1016/j.jhep.2014.12.011. Epub 2014 Dec 13.

Abstract

Background & aims: Anthocyanins may have beneficial effects on lipid metabolism and inflammation and are demonstrated to have hepatoprotective properties in models of restraint-stress- and chemically-induced liver damage. However, their potential to protect against non-alcoholic steatohepatitis (NASH) under conditions relevant for human pathogenesis remains unclear. Therefore, we studied the effects of the standardised anthocyanin-rich extract Mirtoselect on diet-induced NASH in a translational model of disease.

Methods: ApoE(∗)3Leiden mice were fed a Western-type cholesterol-containing diet without (HC) or with 0.1% (w/w) Mirtoselect (HCM) for 20weeks to study the effects on diet-induced NASH.

Results: Mirtoselect attenuated HC-induced hepatic steatosis, as observed by decreased macro- and microvesicular hepatocellular lipid accumulation and reduced hepatic cholesteryl ester content. This anti-steatotic effect was accompanied by local anti-inflammatory effects in liver, as demonstrated by reduced inflammatory cell clusters and reduced neutrophil infiltration in HCM. On a molecular level, HC diet significantly induced hepatic expression of pro-inflammatory genes Tnf, Emr1, Ccl2, Mpo, Cxcl1, and Cxcl2 while this induction was less pronounced or significantly decreased in HCM. A similar quenching effect was observed for HC-induced pro-fibrotic genes, Acta2 and Col1a1 and this anti-fibrotic effect of Mirtoselect was confirmed histologically. Many of the pro-inflammatory and pro-fibrotic parameters positively correlated with intrahepatic free cholesterol levels. Mirtoselect significantly reduced accumulation and crystallisation of intrahepatic free cholesterol, providing a possible mechanism for the observed hepatoprotective effects.

Conclusions: Mirtoselect attenuates development of NASH, reducing hepatic lipid accumulation, inflammation and fibrosis, possibly mediated by local anti-inflammatory effects associated with reduced accumulation and crystallisation of intrahepatic free cholesterol.

Keywords: Anthocyanins; Bilberry; Cholesterol crystals; Fibrosis; Free cholesterol; Non-alcoholic steatohepatitis; Polyphenols; Steatosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Actins / metabolism
  • Animals
  • Anthocyanins / pharmacology*
  • Anti-Infective Agents / pharmacology
  • Calcium-Binding Proteins
  • Chemokine CXCL1 / metabolism
  • Cholesterol Esters / metabolism
  • Cholesterol, Dietary / metabolism
  • Collagen Type I / metabolism
  • Collagen Type I, alpha 1 Chain
  • Cytoprotection
  • Diet, Western
  • Humans
  • Inflammation / drug therapy
  • Inflammation / metabolism
  • Liver / metabolism
  • Liver / pathology
  • Liver Cirrhosis / etiology
  • Liver Cirrhosis / physiopathology
  • Liver Cirrhosis / prevention & control*
  • Mice
  • Non-alcoholic Fatty Liver Disease* / complications
  • Non-alcoholic Fatty Liver Disease* / drug therapy
  • Non-alcoholic Fatty Liver Disease* / physiopathology
  • Plant Extracts
  • Receptors, Cell Surface / metabolism
  • Receptors, G-Protein-Coupled
  • Treatment Outcome
  • Vaccinium myrtillus / chemistry*

Substances

  • Acta2 protein, mouse
  • Actins
  • Adgre1 protein, mouse
  • Anthocyanins
  • Anti-Infective Agents
  • Calcium-Binding Proteins
  • Chemokine CXCL1
  • Cholesterol Esters
  • Cholesterol, Dietary
  • Collagen Type I
  • Collagen Type I, alpha 1 Chain
  • Cxcl1 protein, mouse
  • Plant Extracts
  • Receptors, Cell Surface
  • Receptors, G-Protein-Coupled
  • Vaccinium myrtillus extract