Transient forebrain ischemia produces multiple deficits in dopamine D1 transmission in the lateral neostriatum of the rat

Brain Res. 1989 Oct 2;498(2):376-80. doi: 10.1016/0006-8993(89)91120-7.

Abstract

Striatal dopamine D1 transmission was studied in rats 7 days after transient (30 min) forebrain ischemia using the 4-vessel occlusion model. The striatal distribution of dopamine D1 ([3H]SCH 23390 binding sites) and D2 ([3H]sulpiride binding sites) receptors as well as the distribution of adenylate cyclase ( [3H]forskolin binding sites) and of the intracytoplasmic dopamine and cAMP-regulated phosphoprotein DARPP-32 related to D1 transmission were analyzed. While the distribution of D2 receptors was unaffected 7 days after the ischemic insult, all the other markers showed a patchy disappearance in the dorsolateral part of the neostriatum. These findings underline the existence of selective multiple deficits in D1 transmission after transient forebrain ischemia in rat striatum.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Autoradiography
  • Benzazepines / metabolism
  • Binding Sites
  • Brain Ischemia / metabolism*
  • Colforsin / metabolism
  • Corpus Striatum / metabolism*
  • Diencephalon / blood supply
  • Dopamine Antagonists
  • Dopamine and cAMP-Regulated Phosphoprotein 32
  • Immunohistochemistry
  • Male
  • Nerve Tissue Proteins / metabolism
  • Phosphoproteins / metabolism
  • Rats
  • Rats, Inbred Strains
  • Receptors, Dopamine / physiology*
  • Receptors, Dopamine D1
  • Sulpiride / metabolism
  • Synaptic Transmission*
  • Telencephalon / blood supply

Substances

  • Benzazepines
  • Dopamine Antagonists
  • Dopamine and cAMP-Regulated Phosphoprotein 32
  • Nerve Tissue Proteins
  • Phosphoproteins
  • Receptors, Dopamine
  • Receptors, Dopamine D1
  • Colforsin
  • Sulpiride