Abstract
The arachidonic acid metabolism (AAM) pathway promotes tumour progression. Chemical inhibitors of AAM pathway prolong post-treatment survival of cancer patients. Here we test whether non-synonymous somatic mutations in genes of this pathway, acting as natural inhibitors, increase post-treatment survival. We identify loss-of-function somatic mutations in 15 (18%) of 84 treatment-naïve oral cancer patients by whole-exome sequencing, which we map to genes of AAM pathway. Patients (n = 53) who survived ≥ 12 months after surgery without recurrence have significantly (P = 0.007) higher proportion (26% versus 3%) of mutations than those who did not (n = 31). Patients with mutations have a significantly (P = 0.003) longer median disease-free survival (24 months) than those without (13 months). Compared with the presence of a mutation, absence of any mutation increases the hazard ratio for death (11.3) significantly (P = 0.018). The inferences are strengthened when we pool our data with The Cancer Genome Atlas (TCGA) data. In patients with AAM pathway mutations, some downstream pathways, such as the PI3K-Akt pathway, are downregulated.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Arachidonic Acids / genetics
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Arachidonic Acids / metabolism*
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Carcinoma, Squamous Cell / genetics*
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Carcinoma, Squamous Cell / mortality
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Carcinoma, Squamous Cell / pathology
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Carcinoma, Squamous Cell / surgery
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Cytochrome P-450 CYP2C19 / genetics
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Cytochrome P-450 CYP2C19 / metabolism
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Cytochrome P-450 Enzyme System / genetics
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Cytochrome P-450 Enzyme System / metabolism
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Cytochrome P450 Family 2
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Gene Expression*
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Glutathione Peroxidase
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Group III Phospholipases A2 / genetics
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Group III Phospholipases A2 / metabolism
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Group IV Phospholipases A2 / genetics
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Group IV Phospholipases A2 / metabolism
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Group VI Phospholipases A2 / genetics
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Group VI Phospholipases A2 / metabolism
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Humans
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Metabolic Networks and Pathways / genetics*
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Mouth Neoplasms / genetics*
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Mouth Neoplasms / mortality
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Mouth Neoplasms / pathology
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Mouth Neoplasms / surgery
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Mutation*
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Peroxidases / genetics
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Peroxidases / metabolism
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Proportional Hazards Models
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Survival Analysis
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Thromboxane-A Synthase / genetics
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Thromboxane-A Synthase / metabolism
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Treatment Outcome
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gamma-Glutamyltransferase / genetics
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gamma-Glutamyltransferase / metabolism
Substances
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Arachidonic Acids
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Cytochrome P-450 Enzyme System
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Peroxidases
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GPX7 protein, human
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Glutathione Peroxidase
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CYP2C19 protein, human
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CYP2U1 protein, human
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Cytochrome P-450 CYP2C19
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Cytochrome P450 Family 2
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gamma-Glutamyltransferase
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Group III Phospholipases A2
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Group IV Phospholipases A2
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Group VI Phospholipases A2
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PLA2G3 protein, human
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PLA2G4E protein, human
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PLA2G4F protein, human
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PLA2G6 protein, human
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PTGIS protein, human
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Thromboxane-A Synthase