[Mechanism of the inhibition of proliferation of hapten-modified human and mouse leukemic cells by hapten-specific T-suppressor factor]

Biull Eksp Biol Med. 1989 Jul;108(7):67-9.
[Article in Russian]

Abstract

T-suppressor-inducer and T-suppressor-effector hybridoma supernatants both regulating the DTH reaction intensity triggered by trinitrophenyl-sulphonic acid (TNPSA) or antigen-dependent proliferation of mice lymph node cells to trinitrophenylated bovine serum albumin, possess and ability to inhibit proliferation of TNPSA-modified human and mice leukaemic cells. This effect is specific, and is caused from antigen binding. This effect is specific, and is caused from antigen binding, idiotype positive T-suppressor factors and is based on a cyclic adenosine monophosphate increase of the concentration in target cells.

Publication types

  • Comparative Study
  • English Abstract

MeSH terms

  • Animals
  • Cell Division
  • Cell Line
  • Cyclic AMP / analysis
  • Haptens*
  • Humans
  • Hybridomas
  • Leukemia / immunology*
  • Leukemia / pathology
  • Leukemia, Experimental / immunology*
  • Leukemia, Experimental / pathology
  • Mice
  • T-Lymphocytes, Regulatory / immunology*

Substances

  • Haptens
  • Cyclic AMP