Abstract
Mutations at the K-RAS locus in colon cancer cells are frequently associated with lack of responsiveness to therapy with EGFR inhibitors, as a consequence of the activation of Ras-dependent intracellular signals. Here we report a colon cancer case carrying a novel combination of K-RAS mutations involving codon 13 (Gly to Asp) and codon 19 (Leu to Phe), on separate alleles. The double mutation was restricted to tumor cells bearing a mucinous-like phenotype.
MeSH terms
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Adenocarcinoma, Mucinous / diagnosis*
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Adenocarcinoma, Mucinous / drug therapy
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Adenocarcinoma, Mucinous / pathology
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Aged
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Alleles
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Antibodies, Monoclonal / therapeutic use
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Antibodies, Monoclonal, Humanized / therapeutic use
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Biomarkers, Pharmacological / metabolism
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Biomarkers, Tumor / genetics*
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Cetuximab
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Colonic Neoplasms / diagnosis*
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Colonic Neoplasms / drug therapy
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Colonic Neoplasms / pathology
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DNA Mutational Analysis
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Drug Resistance, Neoplasm / genetics
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Female
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Genes, ras* / genetics
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Humans
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Mutation / genetics*
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Panitumumab
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Protein Kinase Inhibitors / therapeutic use
Substances
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Antibodies, Monoclonal
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Antibodies, Monoclonal, Humanized
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Biomarkers, Pharmacological
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Biomarkers, Tumor
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Protein Kinase Inhibitors
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Panitumumab
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Cetuximab