Cortical superficial siderosis, APOE genotype, and hemorrhage risk in cerebral amyloid angiopathy

Neurology. 2015 Mar 24;84(12):1190-1. doi: 10.1212/WNL.0000000000001406. Epub 2015 Feb 25.

Authors

Prachi Mehndiratta¹, Tadeusz A Mendel²

Affiliations

¹ From the Department of Neurology (P.M.), University of Virginia, Charlottesville; and the Institute of Psychiatry and Neurology (T.A.M.), Second Department of Neurology, Warsaw, Poland. [email protected].
² From the Department of Neurology (P.M.), University of Virginia, Charlottesville; and the Institute of Psychiatry and Neurology (T.A.M.), Second Department of Neurology, Warsaw, Poland.

PMID: 25716363
DOI: 10.1212/WNL.0000000000001406

No abstract available

Publication types

Editorial
Comment

MeSH terms

Apolipoprotein E2 / genetics*
Apolipoprotein E4 / genetics*
Cerebral Amyloid Angiopathy*
Cerebral Cortex / metabolism*
Cerebral Hemorrhage*
Female
Hemosiderosis*
Humans
Male

Substances

Apolipoprotein E2
Apolipoprotein E4