Control of a neuronal morphology program by an RNA-binding zinc finger protein, Unkempt

Genes Dev. 2015 Mar 1;29(5):501-12. doi: 10.1101/gad.258483.115.

Abstract

Cellular morphology is an essential determinant of cellular function in all kingdoms of life, yet little is known about how cell shape is controlled. Here we describe a molecular program that controls the early morphology of neurons through a metazoan-specific zinc finger protein, Unkempt. Depletion of Unkempt in mouse embryos disrupts the shape of migrating neurons, while ectopic expression confers neuronal-like morphology to cells of different nonneuronal lineages. We found that Unkempt is a sequence-specific RNA-binding protein and identified its precise binding sites within coding regions of mRNAs linked to protein metabolism and trafficking. RNA binding is required for Unkempt-induced remodeling of cellular shape and is directly coupled to a reduced production of the encoded proteins. These findings link post-transcriptional regulation of gene expression with cellular shape and have general implications for the development and disease of multicellular organisms.

Keywords: RNA-binding proteins; Unkempt; cell morphology; gene expression program; neurons; translation.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Brain / metabolism
  • Cell Line
  • Cell Shape / genetics*
  • Embryo, Mammalian
  • Gene Expression Profiling
  • Gene Expression Regulation, Developmental*
  • HeLa Cells
  • Humans
  • Mice
  • Neurons / cytology*
  • Protein Binding
  • RNA, Messenger

Substances

  • RNA, Messenger