Long-term administration of prolactin or testosterone induced similar precancerous prostate lesions in rats

Exp Oncol. 2015 Mar;37(1):13-8.

Abstract

Evidence indicates that prolactin plays a crucial role in the normal function and development of the prostate, but abnormal high levels of the hormone are associated with hyperplasia and cancer of the gland.

Aims: The present study was designed to describe the progressive specific histological abnormalities in the prostate of rats with chronic hyperprolactinemia.

Material and methods: Prolactin was administered during 4; 12 or 24 weeks, and the resulting prostatic alterations were compared with control rats, and also with those treated with testosterone, or the combination of prolactin + testosterone.

Results: Rats treated with prolactin, testosterone or prolactin + testosterone expressed precancerous histological abnormalities in the dorsolateral and ventral portions of the prostate as early as in 4 weeks of treatment, but in all cases the malignancy increased after 12 or 24 weeks of treatment.

Conclusion: Our study confirms that chronic hyperprolactinemia is a cause of prostate precancerous pathologies.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Hyperprolactinemia / complications*
  • Hyperprolactinemia / metabolism
  • Male
  • Prolactin / administration & dosage
  • Prolactin / metabolism*
  • Prostate / pathology*
  • Prostatic Neoplasms / etiology*
  • Prostatic Neoplasms / pathology
  • Rats
  • Rats, Wistar
  • Testosterone / administration & dosage
  • Testosterone / metabolism

Substances

  • Testosterone
  • Prolactin