The clinical features of patients with a Y93H variant of hepatitis C virus detected by a PCR invader assay

J Gastroenterol. 2016 Jan;51(1):63-70. doi: 10.1007/s00535-015-1080-1. Epub 2015 Apr 24.

Abstract

Background: Resistance-associated variants (RAVs) reduce the efficacy of interferon (IFN)-free therapy with asunaprevir and daclatasvir for patients infected with hepatitis C virus (HCV) genotype 1b. The characteristics of patients with an L31 or a Y93 variant in the nonstructural 5A region detected by a polymerase chain reaction invader assay were investigated.

Methods: In total, 201 patients with HCV genotype 1b were examined for L31F/M/V variants or a Y93H variant by the polymerase chain reaction invader assay.

Results: L31M and Y93H variants were detected in 4.6 and 21.4 % of patients, respectively. Patients with an L31M variant had no significant characteristics. Patients with a Y93H variant had significantly higher HCV RNA levels (6.5 ± 0.5 log copies per milliliter vs 6.1 ± 0.7 log copies per milliliter, p = 0.0002), higher frequency of mutant type of the IFN-sensitivity-determining region (88.4 % vs 71.7 %, p = 0.0251), and higher frequency of TT genotype at rs8099917 of IL28B (91.7 % vs 54.3 %, p < 0.0001) than those with Y93 wild-type strains. Multivariate analysis identified HCV RNA levels [odds ratio (OR) 3.72, 95 % confidence interval (CI) 1.71-8.06, p = 0.0009] and TT genotype at rs8099917 (OR 7.45, 95 % CI 2.11-26.4, p = 0.0018) as factors associated with the presence of a Y93H variant.

Conclusion: The presence of a Y93H variant was associated with higher HCV RNA levels and TT genotype at rs8099917 of IL28B. Thus, patients with a Y93H variant may be ideal candidates for IFN-based therapy rather than IFN-free therapy, although the high viral load of these patients may reduce the response rate of IFN-based therapy.

Keywords: Direct-acting antiviral agents; Hepatitis C virus; IL28B; PCR invader assay; Resistance-associated variants.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Antiviral Agents / therapeutic use
  • Drug Resistance, Viral / genetics
  • Female
  • Genetic Variation
  • Genotype
  • Hepacivirus / genetics*
  • Hepacivirus / isolation & purification
  • Hepatitis C, Chronic / drug therapy
  • Hepatitis C, Chronic / virology*
  • Humans
  • Interferons / therapeutic use
  • Interleukins / genetics
  • Male
  • Middle Aged
  • Mutation*
  • Polymerase Chain Reaction / methods
  • RNA, Viral / blood
  • Viral Load
  • Viral Nonstructural Proteins / genetics*

Substances

  • Antiviral Agents
  • interferon-lambda, human
  • Interleukins
  • RNA, Viral
  • Viral Nonstructural Proteins
  • Interferons
  • NS-5 protein, hepatitis C virus