Increased expression of IL-28RA mRNA in peripheral blood mononuclear cells from patients with systemic lupus erythematosus

Clin Rheumatol. 2015 Oct;34(10):1807-11. doi: 10.1007/s10067-015-2947-5. Epub 2015 Apr 25.

Abstract

Systemic lupus erythematosus (SLE) is a systemic autoimmune and inflammatory disease with a strong genetic contribution and characterized by kinds of immune reactions. Our previous genome-wide association studies have identified IL-28RA as a susceptibility gene for SLE. In this study, we performed a quantitative reverse transcription polymerase chain reaction (RT-PCR) in 62 patients with SLE and 69 controls to investigate the different expression levels of IL-28RA in peripheral blood mononuclear cells (PBMCs) from SLE patients and healthy controls and the association between IL-28RA expression and systemic lupus erythematosus disease activity index (SLEDAI) or the variant of the single-nucleotide polymorphism (SNP) rs4649203. The expression levels of IL-28RA messenger RNA (mRNA) in SLE patients were significantly increased compared with those of healthy controls. In addition, there were also significant differences in the expression levels of IL-28RA between active (SLEDAI ≥ 6) or inactive (SLEDAI < 6) SLE groups and healthy controls. However, no correlation was observed between IL-28RA mRNA expression level and SLEDAI. There was no association between the variant of the SNP rs4649203 and IL-28RA mRNA expression levels neither. These results indicated that expression of IL-28RA mRNA may be correlated with the pathogenesis of SLE.

Keywords: IL-28RA; Quantitative reverse transcription polymerase chain reaction; Single-nucleotide polymorphism; Systemic lupus erythematosus.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Alleles
  • Case-Control Studies
  • Female
  • Genetic Variation
  • Genotype
  • Humans
  • Leukocytes, Mononuclear / cytology*
  • Lupus Erythematosus, Systemic / blood*
  • Male
  • Middle Aged
  • Polymorphism, Single Nucleotide
  • RNA, Messenger / metabolism
  • Receptors, Cytokine / blood*
  • Receptors, Interferon
  • Reverse Transcriptase Polymerase Chain Reaction

Substances

  • IFNLR1 protein, human
  • RNA, Messenger
  • Receptors, Cytokine
  • Receptors, Interferon