Abstract
CD8(+) T-cell memory phenotype and function are acquired after antigen-driven activation. Memory-like cells may also arise in absence of antigenic exposure in the thymus or in the periphery. Eomesodermin (Eomes) is a key transcription factor for the development of these unconventional memory cells. Herein, we show that type I interferon signalling in CD8(+) T cells directly activates Eomes gene expression. Consistent with this observation, the phenotype, function and age-dependent expansion of 'virtual memory' CD8(+) T cells are strongly affected in absence of type I interferon signalling. In addition, type I interferons induce a sustained expansion of 'virtual memory' CD8(+) T cells in an Eomes-dependent fashion. We further show that the development of 'innate thymic' CD8(+) T cells is dependent on the same pathway. In conclusion, we demonstrate that type I interferon signalling in CD8(+) T cells drives Eomes expression and thereby regulates the function and homeostasis of memory-like CD8(+) T cells.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Animals
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Antigens / metabolism
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CD8-Positive T-Lymphocytes / drug effects
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CD8-Positive T-Lymphocytes / metabolism*
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Gene Expression Regulation / drug effects
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Homeostasis / drug effects
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Immunity, Innate / drug effects
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Immunologic Memory / drug effects
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Immunologic Memory / genetics*
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Interferon Type I / metabolism*
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Interferon-Stimulated Gene Factor 3, gamma Subunit / metabolism
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Interferon-gamma / biosynthesis
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Mice, Inbred BALB C
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Mice, Inbred C57BL
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Poly I-C / pharmacology
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Receptor, Interferon alpha-beta / deficiency
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Receptor, Interferon alpha-beta / metabolism
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Signal Transduction / drug effects
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T-Box Domain Proteins / genetics*
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T-Box Domain Proteins / metabolism
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Thymocytes / drug effects
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Thymocytes / metabolism
Substances
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Antigens
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Eomes protein, mouse
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IRF9 protein, mouse
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Ifnar1 protein, mouse
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Interferon Type I
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Interferon-Stimulated Gene Factor 3, gamma Subunit
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T-Box Domain Proteins
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Receptor, Interferon alpha-beta
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Interferon-gamma
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Poly I-C