Apolipoprotein E*3-Leiden allele results from a partial gene duplication in exon 4

Biochem Biophys Res Commun. 1989 Dec 15;165(2):851-7. doi: 10.1016/s0006-291x(89)80044-0.

Abstract

The apolipoprotein E3-Leiden variant has been shown to be associated with familial dysbetalipoproteinemia (FD) in a dominant manner (Havekes et al., Hum Genet 1986;73:157-163). Applying the polymerase chain reaction technique, we have cloned and sequenced relevant parts of both APOE alleles of the original proband. In exon 4 of the E*3-Leiden allele a partial gene duplication encompassing 21 nucleotides was found, leading to a tandem repeat of the codons 120-126 or 121-127. Using an E3-Leiden mutation specific oligonucleotide probe, the same mutation was found in two additional independently ascertained FD patients with an E3E3 phenotype based on isoelectric focusing. The E*3-Leiden mutation will be useful in the elucidation of the etiology of dominantly inherited forms of FD.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Apolipoprotein E3
  • Apolipoproteins E / blood
  • Apolipoproteins E / genetics*
  • Base Sequence
  • Cloning, Molecular
  • Exons*
  • Genes*
  • Genes, Dominant
  • Genetic Variation*
  • Humans
  • Hyperlipoproteinemia Type III / blood
  • Hyperlipoproteinemia Type III / genetics*
  • Leukocytes / metabolism
  • Molecular Sequence Data
  • Multigene Family*
  • Oligonucleotide Probes
  • Polymerase Chain Reaction
  • Restriction Mapping

Substances

  • Apolipoprotein E3
  • Apolipoproteins E
  • Oligonucleotide Probes
  • apolipoprotein E3 (Leidein)